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Mbio|March 30, 2026
Non-suppressible HIV viremia sustained by clonally expanded CD4+ T cells harboring a genomically defective provirus with an immune-evasive protein expression profileF Harrison Omondi, Yurou Sang, Winnie Dong, et al.American Journal of Nephrology|April 3, 2019
Urinary Biomarkers of Tubular Damage Are Associated with Mortality but Not Cardiovascular Risk among Systolic Blood Pressure Intervention Trial Participants with Chronic Kidney DiseaseVasantha K Jotwani, Alexandra K Lee, Michelle M Estrella, et al.JAMA Network Open|January 28, 2022
Association of Antihypertensives That Stimulate vs Inhibit Types 2 and 4 Angiotensin II Receptors With Cognitive ImpairmentZachary A Marcum, Jordana B Cohen, Chong Zhang, et al.Nature Communications|March 4, 2022
Incorporating temporal distribution of population-level viral load enables real-time estimation of COVID-19 transmissionYun Lin, Bingyi Yang, Sarah Cobey, et al.Alimentary Pharmacology & Therapeutics|June 16, 2016
Liver fibrosis and fatty liver in Asian HIV-infected patientsG Lui, V W-S Wong, G L-H Wong, et al.Cancer|April 27, 2024
Psychological distress and mental health care utilization among Black survivors of adolescent and young adult cancerEunju Choi, Amy M Berkman, Aryce Battle, et al.Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|September 2, 2021
Kidney tubule health, mineral metabolism and adverse events in persons with CKD in SPRINTSimon B Ascher, Rebecca Scherzer, Michelle M Estrella, et al.Gynecologic Oncology|March 24, 2007
HLA-DQB1 polymorphisms and risk for cervical cancer: a case-control study in a southern Chinese populationPaul K S Chan, Jo L K Cheung, Tak-Hong Cheung, et al.Hypertension (Dallas, Tex. : 1979)|August 2, 2017
Visit-to-Visit Office Blood Pressure Variability and Cardiovascular Outcomes in SPRINT (Systolic Blood Pressure Intervention Trial)Tara I Chang, David M Reboussin, Glenn M Chertow, et al.Journal of Molecular Biology|March 6, 2013
Glycan shifting on hepatitis C virus (HCV) E2 glycoprotein is a mechanism for escape from broadly neutralizing antibodiesHomer Pantua, Jingyu Diao, Mark Ultsch, et al.Pageof 222