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K F Shen

Showing results (11-20 of 55) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|November 7, 1995
Ultra-low concentrations of naloxone selectively antagonize excitatory effects of morphine on sensory neurons, thereby increasing its antinociceptive potency and attenuating tolerance/dependence during chronic cotreatmentS M Crain, K F Shen
Brain Research|November 2, 2001
Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependenceK F Shen, S M Crain
Brain Research|August 20, 1990
Cholera toxin-A subunit blocks opioid excitatory effects on sensory neuron action potentials indicating mediation by Gs-linked opioid receptorsK F Shen, S M Crain
Neurochemical Research|November 1, 1996
Modulatory effects of Gs-coupled excitatory opioid receptor functions on opioid analgesia, tolerance, and dependenceS M Crain, K F Shen
Brain Research|October 2, 1995
Chronic morphine-treated sensory ganglion neurons remain supersensitive to the excitatory effects of naloxone for months after return to normal culture medium: an in vitro model of 'protracted opioid dependence'S M Crain, K F Shen
Neuropharmacology|April 1, 1990
Dynorphin prolongs the action potential of mouse sensory ganglion neurons by decreasing a potassium conductance whereas another specific kappa opioid does so by increasing a calcium conductanceK F Shen, S M Crain
Brain Research|November 27, 1992
Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid 'dependence' without toleranceK F Shen, S M Crain
Brain Research|March 13, 1992
After chronic opioid exposure sensory neurons become supersensitive to the excitatory effects of opioid agonists and antagonists as occurs after acute elevation of GM1 gangliosideS M Crain, K F Shen
Brain Research|February 27, 1995
Specific N- or C-terminus modified dynorphin and beta-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonistsK F Shen, S M Crain
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 1, 1994
Nerve growth factor rapidly prolongs the action potential of mature sensory ganglion neurons in culture, and this effect requires activation of Gs-coupled excitatory kappa-opioid receptors on these cellsK F Shen, S M Crain
Pageof 6

Showing results (11-20 of 55) with videos related to

Sort By:
Pageof 6
Proceedings of the National Academy of Sciences of the United States of America|November 7, 1995
Ultra-low concentrations of naloxone selectively antagonize excitatory effects of morphine on sensory neurons, thereby increasing its antinociceptive potency and attenuating tolerance/dependence during chronic cotreatmentS M Crain, K F Shen
Brain Research|November 2, 2001
Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependenceK F Shen, S M Crain
Brain Research|August 20, 1990
Cholera toxin-A subunit blocks opioid excitatory effects on sensory neuron action potentials indicating mediation by Gs-linked opioid receptorsK F Shen, S M Crain
Neurochemical Research|November 1, 1996
Modulatory effects of Gs-coupled excitatory opioid receptor functions on opioid analgesia, tolerance, and dependenceS M Crain, K F Shen
Brain Research|October 2, 1995
Chronic morphine-treated sensory ganglion neurons remain supersensitive to the excitatory effects of naloxone for months after return to normal culture medium: an in vitro model of 'protracted opioid dependence'S M Crain, K F Shen
Neuropharmacology|April 1, 1990
Dynorphin prolongs the action potential of mouse sensory ganglion neurons by decreasing a potassium conductance whereas another specific kappa opioid does so by increasing a calcium conductanceK F Shen, S M Crain
Brain Research|November 27, 1992
Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid 'dependence' without toleranceK F Shen, S M Crain
Brain Research|March 13, 1992
After chronic opioid exposure sensory neurons become supersensitive to the excitatory effects of opioid agonists and antagonists as occurs after acute elevation of GM1 gangliosideS M Crain, K F Shen
Brain Research|February 27, 1995
Specific N- or C-terminus modified dynorphin and beta-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonistsK F Shen, S M Crain
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 1, 1994
Nerve growth factor rapidly prolongs the action potential of mature sensory ganglion neurons in culture, and this effect requires activation of Gs-coupled excitatory kappa-opioid receptors on these cellsK F Shen, S M Crain
Pageof 6