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Current Biology : CB|September 21, 2000
Genetic suppression of phenotypes arising from mutations in dystrophin-related genes in Caenorhabditis elegansK Gieseler, K Grisoni, L SégalatNeurogenetics|February 7, 2001
Dystrobrevin- and dystrophin-like mutants display similar phenotypes in the nematode Caenorhabditis elegansK Gieseler, C Bessou, L SégalatFEBS Letters|November 24, 1999
In vitro interactions of Caenorhabditis elegans dystrophin with dystrobrevin and syntrophinK Gieseler, M Abdel-Dayem, L SégalatZeitschrift Fur Gastroenterologie|January 9, 2023
The Aging Human Liver: The Weal and Woe of Evolutionary LegacyRobert K Gieseler, Thomas Schreiter, Ali CanbayInternational Journal of Molecular Sciences|July 27, 2024
Liver Fibrosis: From Basic Science towards Clinical Progress, Focusing on the Central Role of Hepatic Stellate CellsHikmet Akkız, Robert K Gieseler, Ali CanbayFEBS Letters|December 22, 1999
Mutations in the dystrophin-like dys-1 gene of Caenorhabditis elegans result in reduced acetylcholinesterase activityJ Giugia, K Gieseler, M Arpagaus, et al.Scandinavian Journal of Immunology|December 1, 1993
1,25-Dihydroxyvitamin D3 exerts opposing effects to IL-4 on MHC class-II antigen expression, accessory activity, and phagocytosis of human monocytesH Xu, A Soruri, R K Gieseler, et al.Zeitschrift Fur Gastroenterologie|February 9, 2005
The relationship between apoptosis and non-alcoholic fatty liver disease: an evolutionary cornerstone turned pathogenicA Canbay, R K Gieseler, G J Gores, et al.Journal of Personalized Medicine|May 25, 2024
Liver Diseases: Science, Fiction and the Foreseeable FutureRobert K Gieseler, Theodor Baars, Mustafa K Özçürümez, et al.Metabolism: Clinical and Experimental|March 14, 2022
Towards harnessing the value of organokine crosstalk to predict the risk for cardiovascular disease in non-alcoholic fatty liver diseaseTheodor Baars, Robert K Gieseler, Polykarpos C Patsalis, et al.Pageof 6