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Developmental Medicine and Child Neurology|November 27, 1999
Homozygous factor-V mutation as a genetic cause of perinatal thrombosis and cerebral palsyK H Harum, A H Hoon, G J Kato, et al.Journal of Developmental and Behavioral Pediatrics : JDBP|March 11, 1999
Recent progress in the neurology of learning: memory molecules in the developing brainM V Johnston, K H HarumNeurology|February 13, 2001
Cognitive impairment in Coffin-Lowry syndrome correlates with reduced RSK2 activationK H Harum, L Alemi, M V JohnstonBiochemical and Biophysical Research Communications|May 29, 1987
Cap Z(36/32) is a contaminant and the major inhibitor of actin network formation in conventional actin preparationsJ F Casella, D J MaackThe Journal of Biological Chemistry|March 4, 1994
Interaction of Cap Z with actin. The NH2-terminal domains of the alpha 1 and beta subunits are not required for actin capping, and alpha 1 beta and alpha 2 beta heterodimers bind differentially to actinJ F Casella, M A TorresNucleic Acids Research|December 22, 1999
Members of the SRY family regulate the human LINE retrotransposonsT Tchénio, J F Casella, T HeidmannJournal of Virology|September 1, 1995
Isolation of novel human endogenous retrovirus-like elements with foamy virus-related pol sequenceA Cordonnier, J F Casella, T HeidmannMolecular and Cellular Biology|March 10, 2001
A truncated form of the human CAF-1 p150 subunit impairs the maintenance of transcriptional gene silencing in mammalian cellsT Tchénio, J F Casella, T HeidmannCell Motility and the Cytoskeleton|January 23, 1999
Effect of capping protein, CapZ, on the length of actin filaments and mechanical properties of actin filament networksJ Xu, J F Casella, T D PollardThe Journal of Biological Chemistry|August 15, 1986
Purification and initial characterization of a protein from skeletal muscle that caps the barbed ends of actin filamentsJ F Casella, D J Maack, S LinPageof 7