Showing results (71-80 of 94) with videos related to

Sort By:
Pageof 10
Endocrine|June 1, 1997
Acute, nongenomic actions of the neuroactive gonadal steroid, 3 alpha-hydroxy-4-pregnen-20-one (3 alpha HP), on FSH release in perifused rat anterior pituitary cellsC A Beck, M Wolfe, L D Murphy, et al.
Journal of Steroid Biochemistry|September 1, 1985
Analysis of steroid metabolites produced by theca cells from the adult domestic henB L Marrone, J P Wiebe, K D Buckingham, et al.
American Journal of Respiratory and Critical Care Medicine|March 1, 1996
Noninvasive positive-pressure ventilation in acute respiratory distress without prior chronic respiratory failureW Patrick, K Webster, L Ludwig, et al.
Breast Cancer Research : BCR|May 1, 2015
Progesterone metabolites regulate induction, growth, and suppression of estrogen- and progesterone receptor-negative human breast cell tumorsJohn P Wiebe, Guihua Zhang, Ian Welch, et al.
The Journal of Biological Chemistry|September 5, 1985
The porcine LH/hCG receptor. Characterization and purificationJ Wimalasena, P Moore, J P Wiebe, et al.
Steroids|January 1, 1985
Synthesis of the allylic gonadal steroids, 3 alpha-hydroxy-4-pregnen-20-one and 3 alpha-hydroxy-4-androsten-17-one, and of 3 alpha-hydroxy-5 alpha-pregnan-20-oneJ P Wiebe, C Deline, K D Buckingham, et al.
Journal of Andrology|September 7, 2000
Glycerol disrupts tight junction-associated actin microfilaments, occludin, and microtubules in Sertoli cellsJ P Wiebe, A Kowalik, R L Gallardi, et al.
The Journal of Steroid Biochemistry and Molecular Biology|April 30, 2005
The role of progesterone metabolites in breast cancer: potential for new diagnostics and therapeuticsJ P Wiebe, M J Lewis, V Cialacu, et al.
Child Development|August 23, 2000
Behavior problems among children from different family structures: the influence of genetic self-selectionH H Cleveland, R P Wiebe, E J van den Oord, et al.
The Journal of Steroid Biochemistry and Molecular Biology|January 18, 2015
Progesterone-induced stimulation of mammary tumorigenesis is due to the progesterone metabolite, 5α-dihydroprogesterone (5αP) and can be suppressed by the 5α-reductase inhibitor, finasterideJohn P Wiebe, Martin A Rivas, Maria F Mercogliano, et al.
Pageof 10