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Biochimica Et Biophysica Acta|February 9, 1996
4-Chloro-m-cresol, a potent and specific activator of the skeletal muscle ryanodine receptorA Herrmann-Frank, M Richter, S Sarközi, et al.The Journal of Physiology|August 2, 2000
Malignant hyperthermia mutation Arg615Cys in the porcine ryanodine receptor alters voltage dependence of Ca2+ releaseB Dietze, J Henke, H M Eichinger, et al.Neuroscience Letters|June 19, 2001
Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na+ channel disrupt fast and enhance slow inactivationA K Alekov, W Peter, N Mitrovic, et al.Muscle & Nerve|June 1, 1990
Schwartz-Jampel syndrome: II. Na+ channel defect causes myotoniaF Lehmann-Horn, P A Iaizzo, C Franke, et al.Anasthesiologie, Intensivmedizin, Notfallmedizin, Schmerztherapie : AINS|March 13, 2002
[Anesthesia in neuromuscular disorders. Part 2: specific disorders]C P Baur, U Schara, R Schlecht, et al.Muscle & Nerve|March 1, 1989
Malignant hyperthermia: effects of halothane on the surface membraneP A Iaizzo, F Lehmann-Horn, S R Taylor, et al.Biophysical Journal|January 28, 2003
Voltage-activated calcium signals in myotubes loaded with high concentrations of EGTAR P Schuhmeier, B Dietze, D Ursu, et al.The Journal of Physiology|December 19, 2000
A sodium channel mutation causing epilepsy in man exhibits subtle defects in fast inactivation and activation in vitroA Alekov, M M Rahman, N Mitrovic, et al.Der Anaesthesist|December 1, 1987
[Diagnosis of susceptibility to malignant hyperthermia using the in vitro contracture test]W Klein, C Spiess-Kiefer, G Küther, et al.Brain : a Journal of Neurology|June 4, 1999
Mutant channels contribute <50% to Na+ current in paramyotonia congenita muscleN Mitrovic, A L George, R Rüdel, et al.Pageof 56