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Neurologia I Neurochirurgia Polska|May 1, 1988
[Studies of alanine and aspartate transaminases, lactate dehydrogenase and phosphocreatine kinase levels in the cerebrospinal fluid and serum in multiple sclerosis]K Kos-LisNeurologia I Neurochirurgia Polska|March 1, 1993
[Diabetes and multiple sclerosis]K Kos-Lis, A Wajgt, A Limanska-KwolekProgress in Retinal and Eye Research|July 14, 2019
Axon injury signaling and compartmentalized injury response in glaucomaStephanie B Syc-Mazurek, Richard T LibbyNeurologia I Neurochirurgia Polska|July 1, 1988
[A typical syndrome of the sulco-commissural arteries]K Szyrocka-Szwed, M Maliszewski, B Syc, et al.Cell Death & Disease|July 21, 2017
JUN is important for ocular hypertension-induced retinal ganglion cell degenerationStephanie B Syc-Mazurek, Kimberly A Fernandes, Richard T LibbyCell Death Discovery|October 22, 2019
DDIT3 (CHOP) contributes to retinal ganglion cell somal loss but not axonal degeneration in DBA/2J miceOlivia J Marola, Stephanie B Syc-Mazurek, Richard T LibbyCell Death & Disease|September 27, 2020
Endothelin 1-induced retinal ganglion cell death is largely mediated by JUN activationOlivia J Marola, Stephanie B Syc-Mazurek, Gareth R Howell, et al.Molecular Neurodegeneration|October 4, 2017
Together JUN and DDIT3 (CHOP) control retinal ganglion cell death after axonal injuryStephanie B Syc-Mazurek, Kimberly A Fernandes, Michael P Wilson, et al.Multiple Sclerosis International|March 12, 2013
Quantitative MRI demonstrates abnormality of the fornix and cingulum in multiple sclerosisStephanie B Syc, Daniel M Harrison, Shiv Saidha, et al.Cell Death & Disease|January 27, 2019
Author Correction: Mkk4 and Mkk7 are important for retinal development and axonal injury-induced retinal ganglion cell deathStephanie B Syc-Mazurek, Rebecca L Rausch, Kimberly A Fernandes, et al.Pageof 4