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Proceedings of the National Academy of Sciences of the United States of America|April 29, 1999
The first-nucleotide binding domain of the cystic-fibrosis transmembrane conductance regulator is important for inhibition of the epithelial Na+ channelR Schreiber, A Hopf, M Mall, et al.
EMBO Reports|October 19, 2001
The cystic fibrosis transmembrane conductance regulator (CFTR) inhibits ENaC through an increase in the intracellular Cl- concentrationJ König, R Schreiber, T Voelcker, et al.
The Journal of Membrane Biology|April 12, 2003
Effects of purinergic stimulation, CFTR and osmotic stress on amiloride-sensitive Na+ transport in epithelia and Xenopus oocytesR Schreiber, J König, J Sun, et al.
The Journal of Biological Chemistry|May 13, 1999
Cystic fibrosis transmembrane conductance regulator inhibits epithelial Na+ channels carrying Liddle's syndrome mutationsA Hopf, R Schreiber, M Mall, et al.
Biochemical and Biophysical Research Communications|March 10, 2001
Control of the cystic fibrosis transmembrane conductance regulator by alphaG(i) and RGS proteinsR Schreiber, P Kindle, T Benzing, et al.
Pflugers Archiv : European Journal of Physiology|June 1, 1992
Small-conductance Cl- channels in HT29 cells: activation by Ca2+, hypotonic cell swelling and 8-Br-cGMPK Kunzelmann, R Kubitz, M Grolik, et al.
Human Molecular Genetics|November 13, 1998
Targeted replacement of normal and mutant CFTR sequences in human airway epithelial cells using DNA fragmentsK K Goncz, K Kunzelmann, Z Xu, et al.
Pflugers Archiv : European Journal of Physiology|February 1, 1996
Culture-dependent expression of Na+ conductances in airway epithelial cellsK Kunzelmann, S Kathöfer, A Hipper, et al.
Pflugers Archiv : European Journal of Physiology|September 1, 1993
Small and intermediate conductance chloride channels in HT29 cellsC P Hansen, B Roch, K Kunzelmann, et al.
Pflugers Archiv : European Journal of Physiology|July 1, 1996
cAMP stimulation of CFTR-expressing Xenopus oocytes activates a chromanol-inhibitable K+ conductanceM Mall, K Kunzelmann, A Hipper, et al.
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