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Current Opinion in Cell Biology|February 1, 1996
Contractile protein mutations and heart diseaseK L Vikstrom, L A LeinwandZeitschrift Fur Kardiologie|January 1, 1995
A murine model for hypertrophic cardiomyopathyK L Vikstrom, S M Factor, L A LeinwandMolecular Medicine (Cambridge, Mass.)|September 1, 1996
Mice expressing mutant myosin heavy chains are a model for familial hypertrophic cardiomyopathyK L Vikstrom, S M Factor, L A LeinwandCirculation Research|April 30, 1998
Hypertrophy, pathology, and molecular markers of cardiac pathogenesisK L Vikstrom, T Bohlmeyer, S M Factor, et al.Journal of Molecular and Cellular Cardiology|May 1, 1997
Direct gene transfer into the mouse heartK Li, R E Welikson, K L Vikstrom, et al.Journal of Molecular Biology|February 21, 1997
A 29 residue region of the sarcomeric myosin rod is necessary for filament formationR L Sohn, K L Vikstrom, M Strauss, et al.American Journal of Physiology. Heart and Circulatory Physiology|February 9, 2000
Expression of the beta (slow)-isoform of MHC in the adult mouse heart causes dominant-negative functional effectsJ C Tardiff, T E Hewett, S M Factor, et al.Cell Motility and the Cytoskeleton|January 1, 1993
Sarcomeric myosin heavy chain expressed in nonmuscle cells forms thick filaments in the presence of substoichiometric amounts of light chainsK L Vikstrom, A S Rovner, C G Saez, et al.Cell Structure and Function|February 1, 1997
The vertebrate myosin heavy chain: genetics and assembly propertiesK L Vikstrom, S H Seiler, R L Sohn, et al.Italian Heart Journal : Official Journal of the Italian Federation of Cardiology|August 10, 2000
Modulation of vascular endothelial gene expression by physical training in patients with chronic heart failureM Testa, P V Ennezat, K L Vikstrom, et al.Pageof 10