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Current Opinion in Nephrology and Hypertension|November 1, 1995
Pathogenesis of dialysis-related amyloidosisT Miyata, K MaedaNihon Jinzo Gakkai Shi|January 1, 1992
Effects of excess factor D on early- and late-phase activation of the complement cascadeH Kobayakawa, T Miyata, R Inagi, et al.Biochemical and Biophysical Research Communications|June 30, 1994
Beta 2-microglobulin modified with advanced glycation end products induces interleukin-6 from human macrophages: role in the pathogenesis of hemodialysis-associated amyloidosisY Iida, T Miyata, R Inagi, et al.Nephron|January 1, 1994
Evaluation of the proteolytic activity of factor D accumulated as an active serine protease in patients with chronic renal failureR Inagi, T Miyata, O Oda, et al.Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|February 1, 1997
Oxidation conspires with glycation to generate noxious advanced glycation end products in renal failureT Miyata, K Maeda, K Kurokawa, et al.Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|January 1, 1996
Pathophysiology of advanced glycation end-products in renal failureT Miyata, Y Iida, K Horie, et al.Clinical Immunology and Immunopathology|September 1, 1993
Decreased activity of complement-mediated immune complex clearance in hemodialysis patientsR Inagi, T Miyata, K Hong, et al.Blood|February 15, 1995
Somatic mutations of the PIG-A gene found in Japanese patients with paroxysmal nocturnal hemoglobinuriaN Yamada, T Miyata, K Maeda, et al.Immunology Letters|January 1, 1991
FUT-175 as a potent inhibitor of C5/C3 convertase activity for production of C5a and C3aR Inagi, T Miyata, K Maeda, et al.Kidney International|February 7, 1998
Functional quantitative analysis of the genome in cultured human mesangial cells. Technical noteY Yasuda, T Miyata, M Nangaku, et al.Pageof 216