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K R Chien

Cell

Showing results (1-10 of 6) with videos related to

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Cell|September 18, 1999
Stress pathways and heart failureK R Chien
Cell|April 29, 1999
Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stressH Hirota, J Chen, U A Betz, et al.
Cell|February 7, 1997
MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failureS Arber, J J Hunter, J Ross, et al.
Cell|November 11, 1999
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathyS Minamisawa, M Hoshijima, G Chu, et al.
Cell|December 19, 2001
A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of I(to) and confers susceptibility to ventricular tachycardiaH C Kuo, C F Cheng, R B Clark, et al.
Cell|September 28, 2000
A novel genetic pathway for sudden cardiac death via defects in the transition between ventricular and conduction system cell lineagesV T Nguyên-Trân, S W Kubalak, S Minamisawa, et al.
Pageof 1

Showing results (1-10 of 6) with videos related to

Sort By:
Pageof 1
Cell|September 18, 1999
Stress pathways and heart failureK R Chien
Cell|April 29, 1999
Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stressH Hirota, J Chen, U A Betz, et al.
Cell|February 7, 1997
MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failureS Arber, J J Hunter, J Ross, et al.
Cell|November 11, 1999
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathyS Minamisawa, M Hoshijima, G Chu, et al.
Cell|December 19, 2001
A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of I(to) and confers susceptibility to ventricular tachycardiaH C Kuo, C F Cheng, R B Clark, et al.
Cell|September 28, 2000
A novel genetic pathway for sudden cardiac death via defects in the transition between ventricular and conduction system cell lineagesV T Nguyên-Trân, S W Kubalak, S Minamisawa, et al.
Pageof 1