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K S WOOD

Showing results (21-30 of 59) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|December 1, 1987
Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxideL J Ignarro, G M Buga, K S Wood, et al.
British Journal of Pharmacology|February 1, 1991
Characterization and actions of human umbilical endothelium derived relaxing factorG Chaudhuri, G M Buga, M E Gold, et al.
The American Journal of Physiology|December 1, 1990
L-arginine-dependent vascular smooth muscle relaxation and cGMP formationM E Gold, K S Wood, R E Byrns, et al.
Biochemical and Biophysical Research Communications|June 15, 1989
L-arginine causes whereas L-argininosuccinic acid inhibits endothelium-dependent vascular smooth muscle relaxationM E Gold, K S Wood, G M Buga, et al.
Biology of the Neonate|February 23, 1999
Effect of initial nitric oxide concentration on outcome in infants with persistent pulmonary hypertension of the newbornK S Wood, M J McCaffrey, J C Donovan, et al.
The Journal of Pharmacology and Experimental Therapeutics|March 1, 1984
Association between cyclic GMP accumulation and acetylcholine-elicited relaxation of bovine intrapulmonary arteryL J Ignarro, T M Burke, K S Wood, et al.
Circulation Research|February 1, 1990
Antagonistic modulatory roles of magnesium and calcium on release of endothelium-derived relaxing factor and smooth muscle toneM E Gold, G M Buga, K S Wood, et al.
Biochemical and Biophysical Research Communications|July 31, 1990
Nitric oxide and cyclic GMP formation upon electrical field stimulation cause relaxation of corpus cavernosum smooth muscleL J Ignarro, P A Bush, G M Buga, et al.
Circulation Research|February 1, 1989
Basic polyamino acids rich in arginine, lysine, or ornithine cause both enhancement of and refractoriness to formation of endothelium-derived nitric oxide in pulmonary artery and veinL J Ignarro, M E Gold, G M Buga, et al.
The Journal of Pharmacology and Experimental Therapeutics|June 1, 1985
Differences in responsiveness of intrapulmonary artery and vein to arachidonic acid: mechanism of arterial relaxation involves cyclic guanosine 3':5'-monophosphate and cyclic adenosine 3':5'-monophosphateL J Ignarro, R G Harbison, K S Wood, et al.
Pageof 6

Showing results (21-30 of 59) with videos related to

Sort By:
Pageof 6
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1987
Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxideL J Ignarro, G M Buga, K S Wood, et al.
British Journal of Pharmacology|February 1, 1991
Characterization and actions of human umbilical endothelium derived relaxing factorG Chaudhuri, G M Buga, M E Gold, et al.
The American Journal of Physiology|December 1, 1990
L-arginine-dependent vascular smooth muscle relaxation and cGMP formationM E Gold, K S Wood, R E Byrns, et al.
Biochemical and Biophysical Research Communications|June 15, 1989
L-arginine causes whereas L-argininosuccinic acid inhibits endothelium-dependent vascular smooth muscle relaxationM E Gold, K S Wood, G M Buga, et al.
Biology of the Neonate|February 23, 1999
Effect of initial nitric oxide concentration on outcome in infants with persistent pulmonary hypertension of the newbornK S Wood, M J McCaffrey, J C Donovan, et al.
The Journal of Pharmacology and Experimental Therapeutics|March 1, 1984
Association between cyclic GMP accumulation and acetylcholine-elicited relaxation of bovine intrapulmonary arteryL J Ignarro, T M Burke, K S Wood, et al.
Circulation Research|February 1, 1990
Antagonistic modulatory roles of magnesium and calcium on release of endothelium-derived relaxing factor and smooth muscle toneM E Gold, G M Buga, K S Wood, et al.
Biochemical and Biophysical Research Communications|July 31, 1990
Nitric oxide and cyclic GMP formation upon electrical field stimulation cause relaxation of corpus cavernosum smooth muscleL J Ignarro, P A Bush, G M Buga, et al.
Circulation Research|February 1, 1989
Basic polyamino acids rich in arginine, lysine, or ornithine cause both enhancement of and refractoriness to formation of endothelium-derived nitric oxide in pulmonary artery and veinL J Ignarro, M E Gold, G M Buga, et al.
The Journal of Pharmacology and Experimental Therapeutics|June 1, 1985
Differences in responsiveness of intrapulmonary artery and vein to arachidonic acid: mechanism of arterial relaxation involves cyclic guanosine 3':5'-monophosphate and cyclic adenosine 3':5'-monophosphateL J Ignarro, R G Harbison, K S Wood, et al.
Pageof 6