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Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1984
Size-dependent B lymphocyte subpopulations: relationship of cell volume to surface phenotype, cell cycle, proliferative response, and requirements for antibody production to TNP-Ficoll and TNP-BAC B Thompson, I Scher, M E Schaefer, et al.
Cancer Research|August 12, 1998
"Loop" domain is necessary for taxol-induced mobility shift and phosphorylation of Bcl-2 as well as for inhibiting taxol-induced cytosolic accumulation of cytochrome c and apoptosisG Fang, B S Chang, C N Kim, et al.
Molecular and Cellular Biology|January 7, 1998
Human IAP-like protein regulates programmed cell death downstream of Bcl-xL and cytochrome cC S Duckett, F Li, Y Wang, et al.
The Journal of Biological Chemistry|May 31, 1996
CD30 contains two binding sites with different specificities for members of the tumor necrosis factor receptor-associated factor family of signal transducing proteinsR W Gedrich, M C Gilfillan, C S Duckett, et al.
AJNR. American Journal of Neuroradiology|March 18, 2017
Enhancing Brain Lesions during Acute Optic Neuritis and/or Longitudinally Extensive Transverse Myelitis May Portend a Higher Relapse Rate in Neuromyelitis Optica Spectrum DisordersG Orman, K Y Wang, Y Pekcevik, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 1, 1985
T cell-derived B cell growth factor(s) can induce stimulation of both resting and activated B cellsC B Thompson, M E Schaefer, F D Finkelman, et al.
Cancer Research|June 15, 1995
Bcl-xL is expressed in neuroblastoma cells and modulates chemotherapy-induced apoptosisM G Dole, R Jasty, M J Cooper, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1990
The genomic organization of the CD28 gene. Implications for the regulation of CD28 mRNA expression and heterogeneityK P Lee, C Taylor, B Petryniak, et al.
Blood|August 15, 1996
Bcl-x rather than Bcl-2 mediates CD40-dependent centrocyte survival in the germinal centerJ M Tuscano, K M Druey, A Riva, et al.
The Journal of Experimental Medicine|July 7, 1998
Cytotoxic T lymphocyte antigen 4 (CTLA4) blockade accelerates the acute rejection of cardiac allografts in CD28-deficient mice: CTLA4 can function independently of CD28H Lin, J C Rathmell, G S Gray, et al.
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