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Plos One
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May 23, 2012
A significant but rather mild contribution of T286 autophosphorylation to Ca2+/CaM-stimulated CaMKII activity
Steven J Coultrap, Kelsey Barcomb, K Ulrich Bayer
Biorxiv : the Preprint Server for Biology
|
March 17, 2025
Oxygen/glucose-deprivation causes long-term impairment of synaptic CaMKII movement
Olivia R Buonarati, Nidia Quillinan, K Ulrich Bayer
The EMBO Journal
|
July 12, 2002
Alternative splicing modulates the frequency-dependent response of CaMKII to Ca(2+) oscillations
K Ulrich Bayer, Paul De Koninck, Howard Schulman
Molecular Pharmacology
|
September 24, 2013
Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B
Kelsey Barcomb, Steven J Coultrap, K Ulrich Bayer
Journal of Neurophysiology
|
April 21, 2021
Young DAPK1 knockout mice have altered presynaptic function
Dayton J Goodell, Jonathan E Tullis, K Ulrich Bayer
Journal of Neurophysiology
|
August 19, 2016
Developmental restoration of LTP deficits in heterozygous CaMKIIα KO mice
Dayton J Goodell, Tim A Benke, K Ulrich Bayer
The Journal of Biological Chemistry
|
July 25, 2022
CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity
Sarah G Cook, Nicole L Rumian, K Ulrich Bayer
Journal of Neurochemistry
|
July 28, 2015
Live imaging of endogenous Ca²⁺/calmodulin-dependent protein kinase II in neurons reveals that ischemia-related aggregation does not require kinase activity
Kelsey Barcomb, Dayton J Goodell, Don B Arnold, et al.
Current Opinion in Neurobiology
|
March 31, 2025
CaMKII mechanisms that promote pathological LTP impairments
Matthew E Larsen, Nicole L Rumian, Nidia Quillinan, et al.
The Journal of Biological Chemistry
|
June 2, 2016
The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength
Kelsey Barcomb, Johannes W Hell, Tim A Benke, et al.
Page
of 8
Search research articles
Search
Showing results (11-20 of 77) with videos related to
Sort By:
Page
of 8
Plos One
|
May 23, 2012
A significant but rather mild contribution of T286 autophosphorylation to Ca2+/CaM-stimulated CaMKII activity
Steven J Coultrap, Kelsey Barcomb, K Ulrich Bayer
Biorxiv : the Preprint Server for Biology
|
March 17, 2025
Oxygen/glucose-deprivation causes long-term impairment of synaptic CaMKII movement
Olivia R Buonarati, Nidia Quillinan, K Ulrich Bayer
The EMBO Journal
|
July 12, 2002
Alternative splicing modulates the frequency-dependent response of CaMKII to Ca(2+) oscillations
K Ulrich Bayer, Paul De Koninck, Howard Schulman
Molecular Pharmacology
|
September 24, 2013
Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B
Kelsey Barcomb, Steven J Coultrap, K Ulrich Bayer
Journal of Neurophysiology
|
April 21, 2021
Young DAPK1 knockout mice have altered presynaptic function
Dayton J Goodell, Jonathan E Tullis, K Ulrich Bayer
Journal of Neurophysiology
|
August 19, 2016
Developmental restoration of LTP deficits in heterozygous CaMKIIα KO mice
Dayton J Goodell, Tim A Benke, K Ulrich Bayer
The Journal of Biological Chemistry
|
July 25, 2022
CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity
Sarah G Cook, Nicole L Rumian, K Ulrich Bayer
Journal of Neurochemistry
|
July 28, 2015
Live imaging of endogenous Ca²⁺/calmodulin-dependent protein kinase II in neurons reveals that ischemia-related aggregation does not require kinase activity
Kelsey Barcomb, Dayton J Goodell, Don B Arnold, et al.
Current Opinion in Neurobiology
|
March 31, 2025
CaMKII mechanisms that promote pathological LTP impairments
Matthew E Larsen, Nicole L Rumian, Nidia Quillinan, et al.
The Journal of Biological Chemistry
|
June 2, 2016
The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength
Kelsey Barcomb, Johannes W Hell, Tim A Benke, et al.
Page
of 8