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K Wiegmann

Showing results (11-20 of 32) with videos related to

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The Journal of Biological Chemistry|November 5, 1997
Activation of acid sphingomyelinase by interleukin-1 (IL-1) requires the IL-1 receptor accessory proteinR Hofmeister, K Wiegmann, C Korherr, et al.
The Journal of Biological Chemistry|May 13, 1994
Sphingomyelinase activates proteolytic I kappa B-alpha degradation in a cell-free systemT Machleidt, K Wiegmann, T Henkel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1990
Regulation of IFN-gamma-receptor expression in human monocytes by granulocyte-macrophage colony-stimulating factorT Fischer, K Wiegmann, H Böttinger, et al.
Cell|November 27, 1992
TNF activates NF-kappa B by phosphatidylcholine-specific phospholipase C-induced "acidic" sphingomyelin breakdownS Schütze, K Potthoff, T Machleidt, et al.
The Journal of Biological Chemistry|September 5, 1992
Human 55-kDa receptor for tumor necrosis factor coupled to signal transduction cascadesK Wiegmann, S Schütze, E Kampen, et al.
International Journal of Immunopharmacology|November 1, 1995
Experimental allergic encephalomyelitis, beta-adrenergic receptors and interferon gamma-secreting cells in beta-adrenergic agonist-treated ratsS Muthyala, K Wiegmann, D H Kim, et al.
Journal of Autoimmunity|October 20, 1998
Immunoaugmenting effect of FK 506 on experimental allergic encephalomyelitis in Lewis ratsE Chelmicka-Schorr, K Wiegmann, R Wollmann, et al.
Journal of Neuroimmunology|February 1, 1995
Beta-adrenergic agonists suppress chronic/relapsing experimental allergic encephalomyelitis (CREAE) in Lewis ratsK Wiegmann, S Muthyala, D H Kim, et al.
The Journal of Biological Chemistry|February 20, 1999
Requirement of FADD for tumor necrosis factor-induced activation of acid sphingomyelinaseK Wiegmann, R Schwandner, O Krut, et al.
The Journal of Experimental Medicine|August 1, 1996
Function of the p55 tumor necrosis factor receptor "death domain" mediated by phosphatidylcholine-specific phospholipase CT Machleidt, B Krämer, D Adam, et al.
Pageof 4

Showing results (11-20 of 32) with videos related to

Sort By:
Pageof 4
The Journal of Biological Chemistry|November 5, 1997
Activation of acid sphingomyelinase by interleukin-1 (IL-1) requires the IL-1 receptor accessory proteinR Hofmeister, K Wiegmann, C Korherr, et al.
The Journal of Biological Chemistry|May 13, 1994
Sphingomyelinase activates proteolytic I kappa B-alpha degradation in a cell-free systemT Machleidt, K Wiegmann, T Henkel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1990
Regulation of IFN-gamma-receptor expression in human monocytes by granulocyte-macrophage colony-stimulating factorT Fischer, K Wiegmann, H Böttinger, et al.
Cell|November 27, 1992
TNF activates NF-kappa B by phosphatidylcholine-specific phospholipase C-induced "acidic" sphingomyelin breakdownS Schütze, K Potthoff, T Machleidt, et al.
The Journal of Biological Chemistry|September 5, 1992
Human 55-kDa receptor for tumor necrosis factor coupled to signal transduction cascadesK Wiegmann, S Schütze, E Kampen, et al.
International Journal of Immunopharmacology|November 1, 1995
Experimental allergic encephalomyelitis, beta-adrenergic receptors and interferon gamma-secreting cells in beta-adrenergic agonist-treated ratsS Muthyala, K Wiegmann, D H Kim, et al.
Journal of Autoimmunity|October 20, 1998
Immunoaugmenting effect of FK 506 on experimental allergic encephalomyelitis in Lewis ratsE Chelmicka-Schorr, K Wiegmann, R Wollmann, et al.
Journal of Neuroimmunology|February 1, 1995
Beta-adrenergic agonists suppress chronic/relapsing experimental allergic encephalomyelitis (CREAE) in Lewis ratsK Wiegmann, S Muthyala, D H Kim, et al.
The Journal of Biological Chemistry|February 20, 1999
Requirement of FADD for tumor necrosis factor-induced activation of acid sphingomyelinaseK Wiegmann, R Schwandner, O Krut, et al.
The Journal of Experimental Medicine|August 1, 1996
Function of the p55 tumor necrosis factor receptor "death domain" mediated by phosphatidylcholine-specific phospholipase CT Machleidt, B Krämer, D Adam, et al.
Pageof 4