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Pflugers Archiv : European Journal of Physiology|April 1, 1995
Heterotypic gap junction channels (connexin26-connexin32) violate the paradigm of unitary conductanceF F Bukauskas, C Elfgang, K Willecke, et al.Biophysical Journal|June 1, 1995
Biophysical properties of gap junction channels formed by mouse connexin40 in induced pairs of transfected human HeLa cellsF F Bukauskas, C Elfgang, K Willecke, et al.The EMBO Journal|March 1, 1984
Unstable transformation of mouse 3T3 cells by transfection with DNA from normal human lymphocytesR Schäfer, S Griegel, M A Dubbert, et al.Journal of Cellular Biochemistry|May 1, 1987
The Ha-ras-induced transformed phenotype of rat-1 cells can be suppressed in hybrids with rat embryonic fibroblastsK Willecke, S Griegel, W Martin, et al.European Journal of Cell Biology|June 1, 1992
Molecular cloning of mouse connexins26 and -32: similar genomic organization but distinct promoter sequences of two gap junction genesH Hennemann, G Kozjek, E Dahl, et al.Journal of Cell Science|March 22, 2000
Selective permeability of different connexin channels to the second messenger inositol 1,4,5-trisphosphateH Niessen, H Harz, P Bedner, et al.European Journal of Cell Biology|October 1, 1991
The diversity of connexin genes encoding gap junctional proteinsK Willecke, H Hennemann, E Dahl, et al.The Journal of Membrane Biology|September 1, 1994
Immunochemical characterization of the gap junction protein connexin45 in mouse kidney and transfected human HeLa cellsA Butterweck, U Gergs, C Elfgang, et al.Voprosy Onkologii|January 1, 1985
[Identification of the transforming Ha-ras gene in human melanoma, neuroblastoma and breast cancer cells]P G Kniazev, R Schäfer, K Willecke, et al.Cerebral Cortex (New York, N.Y. : 1991)|July 25, 2000
Myelination defects and neuronal hyperexcitability in the neocortex of connexin 32-deficient miceB Sutor, C Schmolke, B Teubner, et al.Pageof 16