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Circulation Research|January 1, 1997
Hydroxyl radical inhibits sarcoplasmic reticulum Ca(2+)-ATPase function by direct attack on the ATP binding siteK Y Xu, J L Zweier, L C BeckerCirculation Research|July 1, 1995
Functional coupling between glycolysis and sarcoplasmic reticulum Ca2+ transportK Y Xu, J L Zweier, L C BeckerThe Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|May 9, 1998
Ultrastructural localization of glycolytic enzymes on sarcoplasmic reticulum vesticlesK Y Xu, L C BeckerAmerican Journal of Physiology. Heart and Circulatory Physiology|May 18, 2001
Neutrophils are primary source of O2 radicals during reperfusion after prolonged myocardial ischemiaC Duilio, G Ambrosio, P Kuppusamy, et al.Circulation|October 1, 1993
Cardioprotective actions of a monoclonal antibody against CD-18 in myocardial ischemia-reperfusion injuryD J Lefer, S M Shandelya, C V Serrano, et al.The Journal of Pharmacology and Experimental Therapeutics|October 25, 2000
A pyrroline derivative of mexiletine offers marked protection against ischemia/reperfusion-induced myocardial contractile dysfunctionH Li, K Y Xu, L Zhou, et al.The Journal of Biological Chemistry|January 25, 1988
Measurement of superoxide-derived free radicals in the reperfused heart. Evidence for a free radical mechanism of reperfusion injuryJ L ZweierThe Journal of Biological Chemistry|November 25, 1983
Electron paramagnetic resonance measurement of the distance between the metal binding sites of transferrinJ L ZweierBiochimica Et Biophysica Acta|April 17, 1985
Iron-mediated formation of an oxidized adriamycin free radicalJ L ZweierPageof 34