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Cancer Research|November 14, 1997
Betulinic acid triggers CD95 (APO-1/Fas)- and p53-independent apoptosis via activation of caspases in neuroectodermal tumorsS Fulda, C Friesen, M Los, et al.
Oncogene|July 28, 2009
The pyridinylfuranopyrimidine inhibitor, PI-103, chemosensitizes glioblastoma cells for apoptosis by inhibiting DNA repairM-A Westhoff, J A Kandenwein, S Karl, et al.
Cell Death & Disease|January 26, 2013
TRAIL (TNF-related apoptosis-inducing ligand) regulates adipocyte metabolism by caspase-mediated cleavage of PPARgammaM Keuper, I Wernstedt Asterholm, P E Scherer, et al.
Cell Death & Disease|January 19, 2016
Intrinsic and chemo-sensitizing activity of SMAC-mimetics on high-risk childhood acute lymphoblastic leukemiaM Schirmer, L Trentin, M Queudeville, et al.
Leukemia|April 30, 2011
Acute lymphoblastic leukemia cells treated with CpG oligodeoxynucleotides, IL-4 and CD40 ligand facilitate enhanced anti-leukemic CTL responsesD Fabricius, L Breckerbohm, A Vollmer, et al.
Cell Death and Differentiation|November 24, 2007
4-hydroperoxy-cyclophosphamide mediates caspase-independent T-cell apoptosis involving oxidative stress-induced nuclear relocation of mitochondrial apoptogenic factors AIF and EndoGG Strauss, M-A Westhoff, P Fischer-Posovszky, et al.
Oncogene|January 24, 2012
Histone deacetylase inhibitors sensitize glioblastoma cells to TRAIL-induced apoptosis by c-myc-mediated downregulation of cFLIPA Bangert, S Cristofanon, I Eckhardt, et al.
Cell Death and Differentiation|February 14, 2006
Specific resistance upon lentiviral TRAIL transfer by intracellular retention of TRAIL receptorsT Wenger, J Mattern, R Penzel, et al.
Leukemia|December 17, 2011
RIP1 is required for IAP inhibitor-mediated sensitization of childhood acute leukemia cells to chemotherapy-induced apoptosisS Löder, M Fakler, H Schoeneberger, et al.
The Journal of Clinical Endocrinology and Metabolism|December 14, 2011
Loss of enteroendocrine cells in autoimmune-polyendocrine-candidiasis-ectodermal-dystrophy (APECED) syndrome with gastrointestinal dysfunctionC Posovszky, G Lahr, J von Schnurbein, et al.
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