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Leukemia|May 1, 1995
Resistance to APO-1 (CD95) induced apoptosis in T-ALL is determined by a BCL-2 independent anti-apoptotic programK M Debatin, P H KrammerBMC Molecular Biology|September 5, 2001
HIPK2 overexpression leads to stabilization of p53 protein and increased p53 transcriptional activity by decreasing Mdm2 protein levelsY Wang, K M Debatin, H HugJournal of Molecular Medicine (Berlin, Germany)|March 28, 2001
Suicide gene therapy for pediatric tumorsC Beltinger, W Uckert, K M DebatinLeukemia|November 22, 1997
Deficient activation of the CD95 (APO-1/Fas) system in drug-resistant cellsC Friesen, S Fulda, K M DebatinCell Death and Differentiation|April 24, 2004
A critical role of glutathione in determining apoptosis sensitivity and resistance in leukemia cellsC Friesen, Y Kiess, K-M DebatinCell Death and Differentiation|June 25, 1999
Induction of CD95 ligand and apoptosis by doxorubicin is modulated by the redox state in chemosensitive- and drug-resistant tumor cellsC Friesen, S Fulda, K M DebatinPediatric Research|June 1, 1997
CD95-induced apoptosis contributes to loss of primed/memory but not resting/naive T cells in children infected with human immunodeficiency virus type 1T Böhler, S Nedel, K M DebatinGene Therapy|March 29, 2008
Endothelial progenitor cells for cancer gene therapyK-M Debatin, J Wei, C BeltingerClinical and Experimental Immunology|May 3, 2002
Induction of apoptosis and modulation of activation and effector function in T cells by immunosuppressive drugsG Strauss, W Osen, K-M DebatinPageof 20