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Cell Death and Differentiation|June 25, 2025
p53 prophylactic therapy for cancer preventionChristian Krueger, Kanaga SabapathyCell Cycle (Georgetown, Tex.)|December 22, 2004
JNK2: a negative regulator of cellular proliferationKanaga Sabapathy, Erwin F WagnerNeoplasia (New York, N.Y.)|September 28, 2007
Phosphorylation at carboxyl-terminal S373 and S375 residues and 14-3-3 binding are not required for mouse p53 functionMing Kei Lee, Kanaga SabapathyJournal of Cell Science|May 15, 2008
The R246S hot-spot p53 mutant exerts dominant-negative effects in embryonic stem cells in vitro and in vivoMing Kei Lee, Kanaga SabapathyDisease Models & Mechanisms|March 20, 2025
Tumor suppressor genes in the tumor microenvironmentBahareh Tabanifar, Hannah Lau, Kanaga SabapathyCancer Discovery|April 27, 2025
Mutant p53 Gain of Function: Why Many See It, Why Some Do NotGuillermina Lozano, Carol Prives, Kanaga SabapathyThe Journal of Biological Chemistry|May 29, 2015
Interplay between TAp73 Protein and Selected Activator Protein-1 (AP-1) Family Members Promotes AP-1 Target Gene Activation and Cellular GrowthDeepa Subramanian, Wilawan Bunjobpol, Kanaga SabapathySignal Transduction and Targeted Therapy|July 31, 2018
Context-dependent AMPK activation distinctly regulates TAp73 stability and transcriptional activityDan Li, Iqbal Dulloo, Kanaga SabapathyInternational Journal of Molecular Sciences|February 22, 2020
p53-Related Transcription Targets of TAp73 in Cancer Cells-Bona Fide or Distorted Reality?Chao Wang, Cui Rong Teo, Kanaga SabapathyThe Journal of Biological Chemistry|March 1, 2005
Relief of p53-mediated telomerase suppression by p73Wen Hong Toh, Satoru Kyo, Kanaga SabapathyPageof 30