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Hemasphere|January 19, 2026
Highly effective combination of BRG1/BRM inhibitor with BET inhibitor or decitabine for high-risk MECOM-rearranged AMLWarren Fiskus, Christopher P Mill, Jessica Piel, et al.
American Journal of Hematology|October 18, 2018
A phase II study of omacetaxine mepesuccinate for patients with higher-risk myelodysplastic syndrome and chronic myelomonocytic leukemia after failure of hypomethylating agentsNicholas J Short, Elias Jabbour, Kiran Naqvi, et al.
Blood Cancer Journal|March 16, 2025
Superior preclinical efficacy of co-treatment with BRG1/BRM and FLT3 inhibitor against AML cells with FLT3 mutationsWarren Fiskus, Christopher P Mill, Jessica Piel, et al.
Cancer Research|February 8, 2005
Inhibition of glycolysis in cancer cells: a novel strategy to overcome drug resistance associated with mitochondrial respiratory defect and hypoxiaRui-Hua Xu, Helene Pelicano, Yan Zhou, et al.
Surgical Oncology Clinics of North America|September 8, 2004
Neoadjuvant induction chemotherapy followed by chemoradiation: a phase I trial of gemcitabine, cisplatin, and 5-fluorouracil for advanced pancreatic/gastrointestinal malignanciesCharles A Staley, Wayne B Harris, Jerome Landry, et al.
Leukemia|December 22, 2018
Targeting nuclear β-catenin as therapy for post-myeloproliferative neoplasm secondary AMLDyana T Saenz, Warren Fiskus, Taghi Manshouri, et al.
Blood|March 31, 2025
Efficacy of a Novel BCL-xL Degrader, DT2216, in Preclinical Models of JAK2-mutated Post-MPN AMLZhe Wang, Anna Skwarska, Gowri Poigaialwar, et al.
Blood|February 19, 2020
Mechanistic basis and efficacy of targeting the β-catenin-TCF7L2-JMJD6-c-Myc axis to overcome resistance to BET inhibitorsDyana T Saenz, Warren Fiskus, Christopher P Mill, et al.
The Journal of Biological Chemistry|August 23, 2003
Activation of phosphatidylinositol 3-kinase/Akt pathway by androgen through interaction of p85alpha, androgen receptor, and SrcMei Sun, Lin Yang, Richard I Feldman, et al.
Human Molecular Genetics|October 14, 2010
Rapid and transient recruitment of DNMT1 to DNA double-strand breaks is mediated by its interaction with multiple components of the DNA damage response machineryKyungsoo Ha, Gun Eui Lee, Stela S Palii, et al.
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