Showing results (11-20 of 22) with videos related to
Sort By:
Pageof 3
Journal of the American Society of Nephrology : JASN|August 12, 2018
Mechanism of Fibrosis in <i>HNF1B</i>-Related Autosomal Dominant Tubulointerstitial Kidney DiseaseSiu Chiu Chan, Ying Zhang, Annie Shao, et al.Plos One|September 18, 2012
A p53-Pax2 pathway in kidney development: implications for nephrogenesisZubaida Saifudeen, Jiao Liu, Susana Dipp, et al.American Journal of Physiology. Renal Physiology|June 6, 2014
Tissue-specific regulation of the mouse Pkhd1 (ARPKD) gene promoterScott S Williams, Patricia Cobo-Stark, Sachin Hajarnis, et al.Nature Communications|May 28, 2025
An RNA transmethylation pathway governs kidney nephrogenic potentialHarini Ramalingam, Jesus Alvarez, Andrea Flaten, et al.Journal of the American Society of Nephrology : JASN|May 17, 2017
Hepatocyte Nuclear Factor-1<i>β</i> Regulates Urinary Concentration and Response to HypertonicityKaram Aboudehen, Lama Noureddine, Patricia Cobo-Stark, et al.Journal of the American Society of Nephrology : JASN|December 19, 2015
MicroRNA-21 Aggravates Cyst Growth in a Model of Polycystic Kidney DiseaseRonak Lakhia, Sachin Hajarnis, Darren Williams, et al.Journal of the American Society of Nephrology : JASN|December 30, 2015
Transcription Factor Hepatocyte Nuclear Factor-1β Regulates Renal Cholesterol MetabolismKaram Aboudehen, Min Soo Kim, Matthew Mitsche, et al.The Journal of Biological Chemistry|May 3, 2018
Long noncoding RNA <i>Hoxb3os</i> is dysregulated in autosomal dominant polycystic kidney disease and regulates mTOR signalingKaram Aboudehen, Shayan Farahani, Mohammed Kanchwala, et al.JCI Insight|March 18, 2020
Interstitial microRNA miR-214 attenuates inflammation and polycystic kidney disease progressionRonak Lakhia, Matanel Yheskel, Andrea Flaten, et al.Kidney International|June 5, 2017
Loss of transcriptional activation of the potassium channel Kir5.1 by HNF1β drives autosomal dominant tubulointerstitial kidney diseaseAndreas Kompatscher, Jeroen H F de Baaij, Karam Aboudehen, et al.Pageof 3