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Computational and Structural Biotechnology Journal|November 13, 2023
A multi-layered computational structural genomics approach enhances domain-specific interpretation of Kleefstra syndrome variants in EHMT1Young-In Chi, Salomão D Jorge, Davin R Jensen, et al.
American Journal of Respiratory Cell and Molecular Biology|April 28, 2012
NF-κB activation is required for the transition of pulmonary inflammation to muscle atrophyRamon C J Langen, Astrid Haegens, Juanita H J Vernooy, et al.
Cell|October 14, 2004
IKKbeta/NF-kappaB activation causes severe muscle wasting in miceDongsheng Cai, J Daniel Frantz, Nicholas E Tawa, et al.
Bioscience Reports|October 2, 2023
Writers and readers of H3K9me2 form distinct protein networks during the cell cycle that include candidates for H3K9 mimicryGareth Pollin, Thiago M De Assuncao, Salomao Doria Jorge, et al.
Investigative Ophthalmology & Visual Science|December 1, 2012
Retinal not systemic oxidative and inflammatory stress correlated with VEGF expression in rodent models of insulin resistance and diabetesAkira Mima, Weier Qi, Junko Hiraoka-Yamomoto, et al.
Journal of Lipid Research|March 2, 2011
Hepatocyte-specific IKK-β activation enhances VLDL-triglyceride production in APOE*3-Leiden miceJanna A van Diepen, Man C Wong, Bruno Guigas, et al.
Frontiers in Genetics|December 13, 2023
Deep computational phenotyping of genomic variants impacting the SET domain of KMT2C reveal molecular mechanisms for their dysfunctionSalomão Dória Jorge, Young-In Chi, Jose Lizarraga Mazaba, et al.
Computational and Structural Biotechnology Journal|May 26, 2022
Structural bioinformatics enhances the interpretation of somatic mutations in KDM6A found in human cancersYoung-In Chi, Timothy J Stodola, Thiago M De Assuncao, et al.
Cell Metabolism|April 7, 2016
Adipose Natural Killer Cells Regulate Adipose Tissue Macrophages to Promote Insulin Resistance in ObesityByung-Cheol Lee, Myung-Sunny Kim, Munkyong Pae, et al.
Proteins|August 20, 2021
Computational modeling reveals key molecular properties and dynamic behavior of disruptor of telomeric silencing 1-like (DOT1L) and partnering complexes involved in leukemogenesisTimothy J Stodola, Young-In Chi, Thiago M De Assuncao, et al.
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