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Cells|January 16, 2020
Mutations That Confer Drug-Resistance, Oncogenicity and Intrinsic Activity on the ERK MAP Kinases-Current State of the ArtKarina Smorodinsky-Atias, Nadine Soudah, David Engelberg
Proceedings of the National Academy of Sciences of the United States of America|July 8, 2024
Clustered protocadherin cis-interactions are required for combinatorial cell-cell recognition underlying neuronal self-avoidanceGil Wiseglass, Nadir Boni, Karina Smorodinsky-Atias, et al.
Protein Science : a Publication of the Protein Society|December 22, 2025
On the same side: VISTA and its ligands interact in cis on the cell surfaceKarina Smorodinsky-Atias, Gil Wiseglass, Alina Artyukhova, et al.
Oncogene|May 20, 2025
Erk1R84H is an oncoprotein that causes hepatocellular carcinoma in mice and imposes a rigorous negative feedback loopNadine Soudah, Alexey Baskin, Merav Darash-Yahana, et al.
Molecular Biology of the Cell|December 15, 2015
Intrinsically active variants of Erk oncogenically transform cells and disclose unexpected autophosphorylation capability that is independent of TEY phosphorylationKarina Smorodinsky-Atias, Tal Goshen-Lago, Anat Goldberg-Carp, et al.
The Journal of Experimental Medicine|February 20, 2025
HMCN1 variants aggravate epidermolysis bullosa simplex phenotypeShir Bergson, Ofer Sarig, Moshe Giladi, et al.
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