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PLOS Digital Health
|
March 15, 2023
Explainable AI identifies diagnostic cells of genetic AML subtypes
Matthias Hehr, Ario Sadafi, Christian Matek, et al.
Scientific Reports
|
December 1, 2015
Microenvironmental hypoxia regulates FLT3 expression and biology in AML
Silvia Sironi, Michaela Wagner, Alexander Kuett, et al.
Innere Medizin (Heidelberg, Germany)
|
October 1, 2024
[Recurrent hemolysis and iron overload of unclear origin]
Laura Distelmaier, Christian Gebhard, Antje Holzäpfel, et al.
Scientific Data
|
November 11, 2025
A large expert-annotated single-cell peripheral blood dataset for hematological disease diagnostics
Sayedali Shetab Boushehri, Salome Kazeminia, Armin Gruber, et al.
Blood
|
January 1, 2005
FLT3-ITD-TKD dual mutants associated with AML confer resistance to FLT3 PTK inhibitors and cytotoxic agents by overexpression of Bcl-x(L)
Ksenia Bagrintseva, Stefanie Geisenhof, Ruth Kern, et al.
World Neurosurgery
|
April 19, 2019
Perioperative Management of a Patient with Severe Factor V Deficiency Presenting with Chronic Subdural Hematoma: A Clinical Report
Agnes S Meidert, Johannes Kinzinger, Patrick Möhnle, et al.
The American Journal of the Medical Sciences
|
December 14, 2011
Chronic granulomatous disease in an adult recognized by an invasive aspergillosis
Ines Kaufmann, Josef Briegel, Verena van der Heide, et al.
British Journal of Haematology
|
August 24, 2017
Diagnosis of CLL revisited: increased specificity by a modified five-marker scoring system including CD200
Thomas Köhnke, Veronika K Wittmann, Veit L Bücklein, et al.
Blood
|
November 8, 2003
Mutations in the tyrosine kinase domain of FLT3 define a new molecular mechanism of acquired drug resistance to PTK inhibitors in FLT3-ITD-transformed hematopoietic cells
Ksenia Bagrintseva, Ruth Schwab, Tobias M Kohl, et al.
British Journal of Haematology
|
December 6, 2008
Monitoring minimal residual disease in acute myeloid leukaemia with NPM1 mutations by quantitative PCR: clonal evolution is a limiting factor
Christina Papadaki, Annika Dufour, Marlene Seibl, et al.
Page
of 21
Search research articles
Search
Showing results (31-40 of 202) with videos related to
Sort By:
Page
of 21
PLOS Digital Health
|
March 15, 2023
Explainable AI identifies diagnostic cells of genetic AML subtypes
Matthias Hehr, Ario Sadafi, Christian Matek, et al.
Scientific Reports
|
December 1, 2015
Microenvironmental hypoxia regulates FLT3 expression and biology in AML
Silvia Sironi, Michaela Wagner, Alexander Kuett, et al.
Innere Medizin (Heidelberg, Germany)
|
October 1, 2024
[Recurrent hemolysis and iron overload of unclear origin]
Laura Distelmaier, Christian Gebhard, Antje Holzäpfel, et al.
Scientific Data
|
November 11, 2025
A large expert-annotated single-cell peripheral blood dataset for hematological disease diagnostics
Sayedali Shetab Boushehri, Salome Kazeminia, Armin Gruber, et al.
Blood
|
January 1, 2005
FLT3-ITD-TKD dual mutants associated with AML confer resistance to FLT3 PTK inhibitors and cytotoxic agents by overexpression of Bcl-x(L)
Ksenia Bagrintseva, Stefanie Geisenhof, Ruth Kern, et al.
World Neurosurgery
|
April 19, 2019
Perioperative Management of a Patient with Severe Factor V Deficiency Presenting with Chronic Subdural Hematoma: A Clinical Report
Agnes S Meidert, Johannes Kinzinger, Patrick Möhnle, et al.
The American Journal of the Medical Sciences
|
December 14, 2011
Chronic granulomatous disease in an adult recognized by an invasive aspergillosis
Ines Kaufmann, Josef Briegel, Verena van der Heide, et al.
British Journal of Haematology
|
August 24, 2017
Diagnosis of CLL revisited: increased specificity by a modified five-marker scoring system including CD200
Thomas Köhnke, Veronika K Wittmann, Veit L Bücklein, et al.
Blood
|
November 8, 2003
Mutations in the tyrosine kinase domain of FLT3 define a new molecular mechanism of acquired drug resistance to PTK inhibitors in FLT3-ITD-transformed hematopoietic cells
Ksenia Bagrintseva, Ruth Schwab, Tobias M Kohl, et al.
British Journal of Haematology
|
December 6, 2008
Monitoring minimal residual disease in acute myeloid leukaemia with NPM1 mutations by quantitative PCR: clonal evolution is a limiting factor
Christina Papadaki, Annika Dufour, Marlene Seibl, et al.
Page
of 21