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Current Protocols|May 16, 2024
Recombinant Human Proteoglycan Aggrecan-G1 Domain-induced Arthritis (GIA) Mouse ModelKatalin Olasz, Ferenc Boldizsar
Mediators of Inflammation|February 24, 2016
Enigma of IL-17 and Th17 Cells in Rheumatoid Arthritis and in Autoimmune Animal Models of ArthritisReka Kugyelka, Zoltan Kohl, Katalin Olasz, et al.
Cardiovascular Pathology : the Official Journal of the Society for Cardiovascular Pathology|June 12, 2016
FTY720 (Gilenya) treatment prevents spontaneous autoimmune myocarditis and dilated cardiomyopathy in transgenic HLA-DQ8-BALB/c miceFerenc Boldizsar, Oktavia Tarjanyi, Katalin Olasz, et al.
Arthritis and Rheumatism|June 22, 2010
Impaired activation-induced cell death promotes spontaneous arthritis in antigen (cartilage proteoglycan)-specific T cell receptor-transgenic miceFerenc Boldizsar, Katalin Kis-Toth, Oktavia Tarjanyi, et al.
Arthritis and Rheumatism|February 10, 2011
Proteoglycan-induced arthritis and recombinant human proteoglycan aggrecan G1 domain-induced arthritis in BALB/c mice resembling two subtypes of rheumatoid arthritisTibor T Glant, Marianna Radacs, Gyorgy Nagyeri, et al.
Expert Review of Clinical Immunology|May 6, 2010
Immunosenescence and its potential modulation: lessons from mouse modelsFerenc Boldizsar, Katalin Mikecz, Tibor T Glant
International Immunology|March 4, 2009
Th1/Th17 polarization and acquisition of an arthritogenic phenotype in arthritis-susceptible BALB/c, but not in MHC-matched, arthritis-resistant DBA/2 miceFerenc Boldizsar, Oktavia Tarjanyi, Peter Nemeth, et al.
International Journal of Molecular Sciences|April 17, 2025
Proteasome Inhibitors: Potential in Rheumatoid Arthritis Therapy?Oktávia Tarjányi, Katalin Olasz, Fanni Rátky, et al.
Immunity & Ageing : I & A|June 13, 2009
Age-related changes in arthritis susceptibility and severity in a murine model of rheumatoid arthritisOktavia Tarjanyi, Ferenc Boldizsar, Peter Nemeth, et al.
Autoimmunity Reviews|June 19, 2008
Detailed analyses of antibodies recognizing mitochondrial antigens suggest similar or identical mechanism for production of natural antibodies and natural autoantibodiesTamás Czömpöly, Katalin Olasz, Zoltán Nyárády, et al.
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