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Gastroenterology
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September 8, 2015
Human Colon Tumors Express a Dominant-Negative Form of SIGIRR That Promotes Inflammation and Colitis-Associated Colon Cancer in Mice
Junjie Zhao, Katarzyna Bulek, Muhammet F Gulen, et al.
Nature Immunology
|
August 9, 2011
The inducible kinase IKKi is required for IL-17-dependent signaling associated with neutrophilia and pulmonary inflammation
Katarzyna Bulek, Caini Liu, Shadi Swaidani, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
January 27, 2019
IL-17A Recruits Rab35 to IL-17R to Mediate PKCα-Dependent Stress Fiber Formation and Airway Smooth Muscle Contractility
Katarzyna Bulek, Xing Chen, Vandy Parron, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
June 18, 2013
HuR is required for IL-17-induced Act1-mediated CXCL1 and CXCL5 mRNA stabilization
Tomasz Herjan, Peng Yao, Wen Qian, et al.
Nature Communications
|
May 3, 2022
TH17 cells promote CNS inflammation by sensing danger signals via Mincle
Quanri Zhang, Weiwei Liu, Han Wang, et al.
Nature Communications
|
June 1, 2017
IL-17 induced NOTCH1 activation in oligodendrocyte progenitor cells enhances proliferation and inflammatory gene expression
Chenhui Wang, Cun-Jin Zhang, Bradley N Martin, et al.
Immunity
|
April 3, 2007
The Toll-interleukin-1 receptor member SIGIRR regulates colonic epithelial homeostasis, inflammation, and tumorigenesis
Hui Xiao, Muhammet Fatih Gulen, Jinzhong Qin, et al.
Nature Communications
|
February 16, 2020
Inflammation mobilizes copper metabolism to promote colon tumorigenesis via an IL-17-STEAP4-XIAP axis
Yun Liao, Junjie Zhao, Katarzyna Bulek, et al.
The EMBO Journal
|
February 5, 2013
IRAK-M mediates Toll-like receptor/IL-1R-induced NFκB activation and cytokine production
Hao Zhou, Minjia Yu, Koichi Fukuda, et al.
Immunity
|
January 12, 2010
The receptor SIGIRR suppresses Th17 cell proliferation via inhibition of the interleukin-1 receptor pathway and mTOR kinase activation
Muhammet F Gulen, Zizhen Kang, Katarzyna Bulek, et al.
Page
of 4
Search research articles
Search
Showing results (21-30 of 39) with videos related to
Sort By:
Page
of 4
Gastroenterology
|
September 8, 2015
Human Colon Tumors Express a Dominant-Negative Form of SIGIRR That Promotes Inflammation and Colitis-Associated Colon Cancer in Mice
Junjie Zhao, Katarzyna Bulek, Muhammet F Gulen, et al.
Nature Immunology
|
August 9, 2011
The inducible kinase IKKi is required for IL-17-dependent signaling associated with neutrophilia and pulmonary inflammation
Katarzyna Bulek, Caini Liu, Shadi Swaidani, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
January 27, 2019
IL-17A Recruits Rab35 to IL-17R to Mediate PKCα-Dependent Stress Fiber Formation and Airway Smooth Muscle Contractility
Katarzyna Bulek, Xing Chen, Vandy Parron, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
June 18, 2013
HuR is required for IL-17-induced Act1-mediated CXCL1 and CXCL5 mRNA stabilization
Tomasz Herjan, Peng Yao, Wen Qian, et al.
Nature Communications
|
May 3, 2022
TH17 cells promote CNS inflammation by sensing danger signals via Mincle
Quanri Zhang, Weiwei Liu, Han Wang, et al.
Nature Communications
|
June 1, 2017
IL-17 induced NOTCH1 activation in oligodendrocyte progenitor cells enhances proliferation and inflammatory gene expression
Chenhui Wang, Cun-Jin Zhang, Bradley N Martin, et al.
Immunity
|
April 3, 2007
The Toll-interleukin-1 receptor member SIGIRR regulates colonic epithelial homeostasis, inflammation, and tumorigenesis
Hui Xiao, Muhammet Fatih Gulen, Jinzhong Qin, et al.
Nature Communications
|
February 16, 2020
Inflammation mobilizes copper metabolism to promote colon tumorigenesis via an IL-17-STEAP4-XIAP axis
Yun Liao, Junjie Zhao, Katarzyna Bulek, et al.
The EMBO Journal
|
February 5, 2013
IRAK-M mediates Toll-like receptor/IL-1R-induced NFκB activation and cytokine production
Hao Zhou, Minjia Yu, Koichi Fukuda, et al.
Immunity
|
January 12, 2010
The receptor SIGIRR suppresses Th17 cell proliferation via inhibition of the interleukin-1 receptor pathway and mTOR kinase activation
Muhammet F Gulen, Zizhen Kang, Katarzyna Bulek, et al.
Page
of 4