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Kate D Sutherland

Showing results (31-40 of 55) with videos related to

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Developmental Biology|September 9, 2018
Lung morphogenesis is orchestrated through Grainyhead-like 2 (Grhl2) transcriptional programsAriena Kersbergen, Sarah A Best, Sebastian Dworkin, et al.
Cancer Cell|January 15, 2016
Polycomb Repressive Complex 2 Is a Barrier to KRAS-Driven Inflammation and Epithelial-Mesenchymal Transition in Non-Small-Cell Lung CancerMichela Serresi, Gaetano Gargiulo, Natalie Proost, et al.
Cell Death and Differentiation|July 25, 2021
Absence of pro-survival A1 has no impact on inflammatory cell survival in vivo during acute lung inflammation and peritonitisLahiru Gangoda, Robyn L Schenk, Sarah A Best, et al.
Cell Metabolism|March 13, 2018
Synergy between the KEAP1/NRF2 and PI3K Pathways Drives Non-Small-Cell Lung Cancer with an Altered Immune MicroenvironmentSarah A Best, David P De Souza, Ariena Kersbergen, et al.
Journal of Thrombosis and Haemostasis : JTH|March 8, 2025
A glycoprotein VI signaling defect in newly formed platelets generated in stress thrombopoiesisStephanie R Hyslop, Jason Corbin, Pradnya Gangatirkar, et al.
Nature Communications|September 15, 2019
Distinct initiating events underpin the immune and metabolic heterogeneity of KRAS-mutant lung adenocarcinomaSarah A Best, Sheryl Ding, Ariena Kersbergen, et al.
Nature Cell Biology|December 26, 2006
Gata-3 is an essential regulator of mammary-gland morphogenesis and luminal-cell differentiationMarie-Liesse Asselin-Labat, Kate D Sutherland, Holly Barker, et al.
The EMBO Journal|December 2, 2006
c-myc as a mediator of accelerated apoptosis and involution in mammary glands lacking Socs3Kate D Sutherland, François Vaillant, Warren S Alexander, et al.
Oncogene|June 15, 2020
Targeting platelets for improved outcome in KRAS-driven lung adenocarcinomaStephanie R Hyslop, Marliese Alexander, Alesha A Thai, et al.
Biorxiv : the Preprint Server for Biology|October 1, 2025
SMARCA4 is essential for early-stage tumor development but its loss promotes late-stage cancer progression in small-cell lung cancerNicole A Kirk, Jin Ng, Kate-Lin Ly, et al.
Pageof 6

Showing results (31-40 of 55) with videos related to

Sort By:
Pageof 6
Developmental Biology|September 9, 2018
Lung morphogenesis is orchestrated through Grainyhead-like 2 (Grhl2) transcriptional programsAriena Kersbergen, Sarah A Best, Sebastian Dworkin, et al.
Cancer Cell|January 15, 2016
Polycomb Repressive Complex 2 Is a Barrier to KRAS-Driven Inflammation and Epithelial-Mesenchymal Transition in Non-Small-Cell Lung CancerMichela Serresi, Gaetano Gargiulo, Natalie Proost, et al.
Cell Death and Differentiation|July 25, 2021
Absence of pro-survival A1 has no impact on inflammatory cell survival in vivo during acute lung inflammation and peritonitisLahiru Gangoda, Robyn L Schenk, Sarah A Best, et al.
Cell Metabolism|March 13, 2018
Synergy between the KEAP1/NRF2 and PI3K Pathways Drives Non-Small-Cell Lung Cancer with an Altered Immune MicroenvironmentSarah A Best, David P De Souza, Ariena Kersbergen, et al.
Journal of Thrombosis and Haemostasis : JTH|March 8, 2025
A glycoprotein VI signaling defect in newly formed platelets generated in stress thrombopoiesisStephanie R Hyslop, Jason Corbin, Pradnya Gangatirkar, et al.
Nature Communications|September 15, 2019
Distinct initiating events underpin the immune and metabolic heterogeneity of KRAS-mutant lung adenocarcinomaSarah A Best, Sheryl Ding, Ariena Kersbergen, et al.
Nature Cell Biology|December 26, 2006
Gata-3 is an essential regulator of mammary-gland morphogenesis and luminal-cell differentiationMarie-Liesse Asselin-Labat, Kate D Sutherland, Holly Barker, et al.
The EMBO Journal|December 2, 2006
c-myc as a mediator of accelerated apoptosis and involution in mammary glands lacking Socs3Kate D Sutherland, François Vaillant, Warren S Alexander, et al.
Oncogene|June 15, 2020
Targeting platelets for improved outcome in KRAS-driven lung adenocarcinomaStephanie R Hyslop, Marliese Alexander, Alesha A Thai, et al.
Biorxiv : the Preprint Server for Biology|October 1, 2025
SMARCA4 is essential for early-stage tumor development but its loss promotes late-stage cancer progression in small-cell lung cancerNicole A Kirk, Jin Ng, Kate-Lin Ly, et al.
Pageof 6