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Journal of Autoimmunity|September 18, 2012
Recombinant IL-6 treatment protects mice from organ specific autoimmune disease by IL-6 classical signalling-dependent IL-1ra inductionUnni Krishna S R L Samavedam, Kathrin Kalies, Jürgen Scheller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 21, 2013
B cells, dendritic cells, and macrophages are required to induce an autoreactive CD4 helper T cell response in experimental epidermolysis bullosa acquisitaHiroaki Iwata, Katja Bieber, Benjamin Tiburzy, et al.
European Journal of Immunology|October 12, 2013
Activated CD4+ T cells enter the splenic T-cell zone and induce autoantibody-producing germinal centers through bystander activationDavid Banczyk, Kathrin Kalies, Lars Nachbar, et al.
The Journal of Investigative Dermatology|May 26, 2020
Propranolol Is an Effective Topical and Systemic Treatment Option for Experimental Epidermolysis Bullosa AcquisitaPia Stüssel, Katharina Schulze Dieckhoff, Sven Künzel, et al.
Frontiers in Immunology|December 12, 2017
Regulatory T Cells Suppress Inflammation and Blistering in Pemphigoid DiseasesKatja Bieber, Shijie Sun, Mareike Witte, et al.
Molecular Medicine (Cambridge, Mass.)|December 22, 2016
Reduced skin blistering in experimental epidermolysis bullosa acquisita after anti-TNF treatmentMisa Hirose, Anika Kasprick, Foteini Beltsiou, et al.
Gastroenterology|July 23, 2013
Mitochondrial gene polymorphisms that protect mice from colitisFlorian Bär, Wiebke Bochmann, Andrea Widok, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|July 16, 2017
Circadian rhythm disruption impairs tissue homeostasis and exacerbates chronic inflammation in the intestineRené Pagel, Florian Bär, Torsten Schröder, et al.
The Journal of Pathology|May 9, 2015
The retinoid-related orphan receptor alpha is essential for the end-stage effector phase of experimental epidermolysis bullosa acquisitaHengameh Sadeghi, Yask Gupta, Steffen Möller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 22, 2015
Caspase-1-independent IL-1 release mediates blister formation in autoantibody-induced tissue injury through modulation of endothelial adhesion moleculesHengameh Sadeghi, Anike Lockmann, Anna-Carina Hund, et al.
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