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Molecular & Cellular Proteomics : MCP|September 20, 2018
Chromobodies to Quantify Changes of Endogenous Protein Concentration in Living CellsBettina-Maria Keller, Julia Maier, Kathy-Ann Secker, et al.
Cancers|June 11, 2020
Only Hematopoietic Stem and Progenitor Cells from Cord Blood Are Susceptible to Malignant Transformation by <i>MLL-AF4</i> TranslocationsKathy-Ann Secker, Lukas Bruns, Hildegard Keppeler, et al.
Frontiers in Immunology|August 22, 2018
Culture-Expanded Human Invariant Natural Killer T Cells Suppress T-Cell Alloreactivity and Eradicate LeukemiaHannes Schmid, Corina Schneidawind, Simona Jahnke, et al.
Oncogene|August 17, 2019
Inhibition of DOT1L and PRMT5 promote synergistic anti-tumor activity in a human MLL leukemia model induced by CRISPR/Cas9Kathy-Ann Secker, Hildegard Keppeler, Silke Duerr-Stoerzer, et al.
Cancers|May 28, 2020
MAT2A as Key Regulator and Therapeutic Target in <i>MLL</i>r LeukemogenesisKathy-Ann Secker, Bianca Bloechl, Hildegard Keppeler, et al.
Arthritis Research & Therapy|April 2, 2020
Inhibition of effector B cells by ibrutinib in systemic sclerosisJakob Einhaus, Ann-Christin Pecher, Elisa Asteriti, et al.
Neoplasia (New York, N.Y.)|May 6, 2023
Targeting MYC in combination with epigenetic regulators induces synergistic anti-leukemic effects in MLLr leukemia and simultaneously improves immunityRahel Fitzel, Kathy-Ann Secker-Grob, Hildegard Keppeler, et al.
Haematologica|January 14, 2021
Human invariant natural killer T cells promote tolerance by preferential apoptosis induction of conventional dendritic cellsHannes Schmid, Emmanuelle M Ribeiro, Kathy-Ann Secker, et al.
Oncogene|November 29, 2023
The RORɣ/SREBP2 pathway is a master regulator of cholesterol metabolism and serves as potential therapeutic target in t(4;11) leukemiaEstelle Erkner, Thomas Hentrich, Rebekka Schairer, et al.
Arthritis Research & Therapy|October 17, 2019
Invariant natural killer T cells are functionally impaired in patients with systemic sclerosisAnn-Christin Pecher, Felix Kettemann, Elisa Asteriti, et al.
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