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Experimental Animals|November 2, 2011
Transmitochondrial mice as models for mitochondrial DNA-based diseasesKazuto Nakada, Jun-Ichi HayashiRinsho Shinkeigaku = Clinical Neurology|February 10, 2009
[Model mouse for mitochondrial DNA-based diseases]Kazuto Nakada, Akitsugu Sato, Jun-Ichi HayashiProceedings of the Japan Academy. Series B, Physical and Biological Sciences|October 23, 2008
Reverse genetic studies of mitochondrial DNA-based diseases using a mouse modelKazuto Nakada, Akitsugu Sato, Jun-Ichi HayashiBiofactors (Oxford, England)|May 19, 2009
Mitochondrial complementation preventing respiratory dysfunction caused by mutant mtDNAAkitsugu Sato, Kazuto Nakada, Jun-Ichi HayashiBiochimica Et Biophysica Acta|April 21, 2006
Mitochondrial dynamics and aging: Mitochondrial interaction preventing individuals from expression of respiratory deficiency caused by mutant mtDNAAkitsugu Sato, Kazuto Nakada, Jun-Ichi HayashiHuman Molecular Genetics|June 12, 2010
Lactic acidemia in the pathogenesis of mice carrying mitochondrial DNA with a deletionEmi Ogasawara, Kazuto Nakada, Jun-Ichi HayashiThe International Journal of Biochemistry & Cell Biology|May 26, 2009
Mitochondrial functional complementation in mitochondrial DNA-based diseasesKazuto Nakada, Akitsugu Sato, Jun-ichi HayashiMitochondrion|August 27, 2005
A novel defense system of mitochondria in mice and human subjects for preventing expression of mitochondrial dysfunction by pathogenic mutant mtDNAsKazuto Nakada, Tomoko Ono, Jun-Ichi HayashiBiochemical and Biophysical Research Communications|January 31, 2004
Presence of interaction but not complementation between human mtDNAs carrying different mutations within a tRNA(Leu(UUR)) geneTomoko Ono, Yoshiko Kasahara, Kazuto Nakada, et al.G3 (Bethesda, Md.)|July 16, 2013
Mitochondrial DNA with a large-scale deletion causes two distinct mitochondrial disease phenotypes in miceShun Katada, Takayuki Mito, Emi Ogasawara, et al.Pageof 15