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Scientific Reports|February 23, 2012
Zic2 hypomorphic mutant mice as a schizophrenia model and ZIC2 mutations identified in schizophrenia patientsMinoru Hatayama, Akira Ishiguro, Yoshimi Iwayama, et al.Histopathology|March 26, 2018
Low-grade intraductal carcinoma of the salivary gland with prominent oncocytic change: a newly described variantMasato Nakaguro, Makoto Urano, Hiroaki Suzuki, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 5, 2002
Delphilin: a novel PDZ and formin homology domain-containing protein that synaptically colocalizes and interacts with glutamate receptor delta 2 subunitYohei Miyagi, Tetsuji Yamashita, Masahiro Fukaya, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 5, 2007
Extracellular signal-regulated kinase 2 (ERK2) knockdown mice show deficits in long-term memory; ERK2 has a specific function in learning and memoryYasushi Satoh, Shogo Endo, Toshio Ikeda, et al.Neuroscience Research|February 27, 2018
Loss of GPRC5B impairs synapse formation of Purkinje cells with cerebellar nuclear neurons and disrupts cerebellar synaptic plasticity and motor learningTakamitsu Sano, Ayako Kohyama-Koganeya, Masami O Kinoshita, et al.Nature Neuroscience|July 5, 2011
Potent amyloidogenicity and pathogenicity of Aβ43Takashi Saito, Takahiro Suemoto, Nathalie Brouwers, et al.Iscience|July 5, 2022
Slitrk2 deficiency causes hyperactivity with altered vestibular function and serotonergic dysregulationKei-Ichi Katayama, Naoko Morimura, Katsunori Kobayashi, et al.Neurobiology of Disease|September 8, 2006
Loss of M5 muscarinic acetylcholine receptors leads to cerebrovascular and neuronal abnormalities and cognitive deficits in miceRuna Araya, Takanori Noguchi, Munehiro Yuhki, et al.Nature Communications|June 13, 2017
Autism-like behaviours and enhanced memory formation and synaptic plasticity in Lrfn2/SALM1-deficient miceNaoko Morimura, Hiroki Yasuda, Kazuhiko Yamaguchi, et al.Experimental Neurology|March 25, 2017
Ts1Cje Down syndrome model mice exhibit environmental stimuli-triggered locomotor hyperactivity and sociability concurrent with increased flux through central dopamine and serotonin metabolismAtsushi Shimohata, Keiichi Ishihara, Satoko Hattori, et al.Pageof 5