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Ke Cong

Showing results (11-20 of 21) with videos related to

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Biorxiv : the Preprint Server for Biology|January 23, 2024
FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cellsKe Cong, Nathan MacGilvary, Silviana Lee, et al.
Nature Communications|March 24, 2024
FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cellsKe Cong, Nathan MacGilvary, Silviana Lee, et al.
Molecular Cell|April 17, 2026
MRE11 proximal polyadenylation site-mediated looping impacts transcription and genomic stabilityKaimeng Huang, Marie Eve Brault, Ke Cong, et al.
Science Advances|June 25, 2020
Inhibition of the translesion synthesis polymerase REV1 exploits replication gaps as a cancer vulnerabilitySumeet Nayak, Jennifer A Calvo, Ke Cong, et al.
Cell Reports|September 21, 2018
Opposing Roles of FANCJ and HLTF Protect Forks and Restrain Replication during StressMin Peng, Ke Cong, Nicholas J Panzarino, et al.
Molecular Cell|September 11, 2025
RAD51 is chromatin enriched and targetable in BRCA1-deficient cellsMin Peng, Silviana Lee, Hitha Gopalan Nair, et al.
Cancer Research|November 13, 2020
Replication Gaps Underlie BRCA Deficiency and Therapy ResponseNicholas J Panzarino, John J Krais, Ke Cong, et al.
Molecular Cell|July 3, 2021
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiencyKe Cong, Min Peng, Arne Nedergaard Kousholt, et al.
Molecular Cell|August 6, 2021
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiencyKe Cong, Min Peng, Arne Nedergaard Kousholt, et al.
Molecular Cell|September 23, 2021
Loss of nuclear DNA ligase III reverts PARP inhibitor resistance in BRCA1/53BP1 double-deficient cells by exposing ssDNA gapsMariana Paes Dias, Vivek Tripathi, Ingrid van der Heijden, et al.
Pageof 3

Showing results (11-20 of 21) with videos related to

Sort By:
Pageof 3
Biorxiv : the Preprint Server for Biology|January 23, 2024
FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cellsKe Cong, Nathan MacGilvary, Silviana Lee, et al.
Nature Communications|March 24, 2024
FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cellsKe Cong, Nathan MacGilvary, Silviana Lee, et al.
Molecular Cell|April 17, 2026
MRE11 proximal polyadenylation site-mediated looping impacts transcription and genomic stabilityKaimeng Huang, Marie Eve Brault, Ke Cong, et al.
Science Advances|June 25, 2020
Inhibition of the translesion synthesis polymerase REV1 exploits replication gaps as a cancer vulnerabilitySumeet Nayak, Jennifer A Calvo, Ke Cong, et al.
Cell Reports|September 21, 2018
Opposing Roles of FANCJ and HLTF Protect Forks and Restrain Replication during StressMin Peng, Ke Cong, Nicholas J Panzarino, et al.
Molecular Cell|September 11, 2025
RAD51 is chromatin enriched and targetable in BRCA1-deficient cellsMin Peng, Silviana Lee, Hitha Gopalan Nair, et al.
Cancer Research|November 13, 2020
Replication Gaps Underlie BRCA Deficiency and Therapy ResponseNicholas J Panzarino, John J Krais, Ke Cong, et al.
Molecular Cell|July 3, 2021
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiencyKe Cong, Min Peng, Arne Nedergaard Kousholt, et al.
Molecular Cell|August 6, 2021
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiencyKe Cong, Min Peng, Arne Nedergaard Kousholt, et al.
Molecular Cell|September 23, 2021
Loss of nuclear DNA ligase III reverts PARP inhibitor resistance in BRCA1/53BP1 double-deficient cells by exposing ssDNA gapsMariana Paes Dias, Vivek Tripathi, Ingrid van der Heijden, et al.
Pageof 3