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Keigyou Yoh

Showing results (1-10 of 56) with videos related to

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Nihon Rinsho. Japanese Journal of Clinical Medicine|March 10, 2006
[Methodology in molecular nephrology]Keigyou Yoh, Akio Koyama
Experimental Animals|July 10, 2015
Th2-biased GATA-3 transgenic mice developed severe experimental peritoneal fibrosis compared with Th1-biased T-bet and Th17-biased RORγt transgenic miceKeigyou Yoh, Masami Ojima, Satoru Takahashi
Experimental Animals|April 29, 2014
Overexpression of GATA-3 in T cells accelerates dextran sulfate sodium-induced colitisMidori Okamura, Keigyou Yoh, Masami Ojima, et al.
Biochemical and Biophysical Research Communications|June 20, 2006
MafK overexpression in pancreatic beta-cells caused impairment of glucose-stimulated insulin secretionHomare Shimohata, Keigyou Yoh, Naoki Morito, et al.
Oncogene|December 19, 2003
Nrf2 regulates the sensitivity of death receptor signals by affecting intracellular glutathione levelsNaoki Morito, Keigyou Yoh, Ken Itoh, et al.
Frontiers in Immunology|November 23, 2017
Differentiation of IL-17-Producing Invariant Natural Killer T Cells Requires Expression of the Transcription Factor c-MafJhang-Sian Yu, Michito Hamada, Shigeo Ohtsuka, et al.
Journal of Nephrology|July 14, 2005
Staphylococcal cell membrane antigen, a possible antigen in post-methicillin resistant Staphylococcus aureus (MRSA) infection nephritis and IgA nephropathy, exhibits high immunogenic activity that is enhanced by superantigenYoshio Shimizu, Hideko Sakurai, Kouichi Hirayama, et al.
Journal of Nephrology|February 21, 2009
Overexpression of T-bet in T cells accelerates autoimmune glomerulonephritis in mice with a dominant Th1 backgroundHomare Shimohata, Akiko Yamada, Keigyou Yoh, et al.
Journal of Nephrology|October 8, 2009
Hemizygous Fabry disease associated with IgA nephropathy: a case reportHomare Shimohata, Keigyou Yoh, Kenji Takada, et al.
Brain, Behavior, and Immunity|July 22, 2014
Th17-biased RORγt transgenic mice become susceptible to a viral model for multiple sclerosisNicholas E Martinez, Fumitaka Sato, Eiichiro Kawai, et al.
Pageof 6

Showing results (1-10 of 56) with videos related to

Sort By:
Pageof 6
Nihon Rinsho. Japanese Journal of Clinical Medicine|March 10, 2006
[Methodology in molecular nephrology]Keigyou Yoh, Akio Koyama
Experimental Animals|July 10, 2015
Th2-biased GATA-3 transgenic mice developed severe experimental peritoneal fibrosis compared with Th1-biased T-bet and Th17-biased RORγt transgenic miceKeigyou Yoh, Masami Ojima, Satoru Takahashi
Experimental Animals|April 29, 2014
Overexpression of GATA-3 in T cells accelerates dextran sulfate sodium-induced colitisMidori Okamura, Keigyou Yoh, Masami Ojima, et al.
Biochemical and Biophysical Research Communications|June 20, 2006
MafK overexpression in pancreatic beta-cells caused impairment of glucose-stimulated insulin secretionHomare Shimohata, Keigyou Yoh, Naoki Morito, et al.
Oncogene|December 19, 2003
Nrf2 regulates the sensitivity of death receptor signals by affecting intracellular glutathione levelsNaoki Morito, Keigyou Yoh, Ken Itoh, et al.
Frontiers in Immunology|November 23, 2017
Differentiation of IL-17-Producing Invariant Natural Killer T Cells Requires Expression of the Transcription Factor c-MafJhang-Sian Yu, Michito Hamada, Shigeo Ohtsuka, et al.
Journal of Nephrology|July 14, 2005
Staphylococcal cell membrane antigen, a possible antigen in post-methicillin resistant Staphylococcus aureus (MRSA) infection nephritis and IgA nephropathy, exhibits high immunogenic activity that is enhanced by superantigenYoshio Shimizu, Hideko Sakurai, Kouichi Hirayama, et al.
Journal of Nephrology|February 21, 2009
Overexpression of T-bet in T cells accelerates autoimmune glomerulonephritis in mice with a dominant Th1 backgroundHomare Shimohata, Akiko Yamada, Keigyou Yoh, et al.
Journal of Nephrology|October 8, 2009
Hemizygous Fabry disease associated with IgA nephropathy: a case reportHomare Shimohata, Keigyou Yoh, Kenji Takada, et al.
Brain, Behavior, and Immunity|July 22, 2014
Th17-biased RORγt transgenic mice become susceptible to a viral model for multiple sclerosisNicholas E Martinez, Fumitaka Sato, Eiichiro Kawai, et al.
Pageof 6