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Keith W Caldecott

Showing results (91-100 of 116) with videos related to

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Neurology. Genetics|May 2, 2019
Novel PNKP mutations causing defective DNA strand break repair and PARP1 hyperactivity in MCSZIlona Kalasova, Hana Hanzlikova, Neerja Gupta, et al.
Nucleic Acids Research|September 20, 2013
PARP-1 dependent recruitment of the amyotrophic lateral sclerosis-associated protein FUS/TLS to sites of oxidative DNA damageStuart L Rulten, Amy Rotheray, Ryan L Green, et al.
Neurology. Genetics|August 16, 2018
Confirming TDP2 mutation in spinocerebellar ataxia autosomal recessive 23 (SCAR23)Guido Zagnoli-Vieira, Francesco Bruni, Kyle Thompson, et al.
Nucleic Acids Research|June 29, 2012
TDP2 promotes repair of topoisomerase I-mediated DNA damage in the absence of TDP1Zhihong Zeng, Abhishek Sharma, Limei Ju, et al.
Cell Reports|January 17, 2019
Efficient Single-Strand Break Repair Requires Binding to Both Poly(ADP-Ribose) and DNA by the Central BRCT Domain of XRCC1Luis M Polo, Yingqi Xu, Peter Hornyak, et al.
Molecular Cell|January 8, 2011
PARP-3 and APLF function together to accelerate nonhomologous end-joiningStuart L Rulten, Anna E O Fisher, Isabelle Robert, et al.
Life Science Alliance|February 28, 2019
FUS (fused in sarcoma) is a component of the cellular response to topoisomerase I-induced DNA breakage and transcriptional stressMaria Isabel Martinez-Macias, Duncan Aq Moore, Ryan L Green, et al.
EMBO Reports|May 1, 2021
Parp1 hyperactivity couples DNA breaks to aberrant neuronal calcium signalling and lethal seizuresEmilia Komulainen, Jack Badman, Stephanie Rey, et al.
Nature|September 3, 2025
DNA2 enables growth by restricting recombination-restarted replicationJessica J R Hudson, Rowin Appanah, David Jones, et al.
DNA Repair|September 24, 2004
The ataxia-oculomotor apraxia 1 gene product has a role distinct from ATM and interacts with the DNA strand break repair proteins XRCC1 and XRCC4Paula M Clements, Claire Breslin, Emma D Deeks, et al.
Pageof 12

Showing results (91-100 of 116) with videos related to

Sort By:
Pageof 12
Neurology. Genetics|May 2, 2019
Novel PNKP mutations causing defective DNA strand break repair and PARP1 hyperactivity in MCSZIlona Kalasova, Hana Hanzlikova, Neerja Gupta, et al.
Nucleic Acids Research|September 20, 2013
PARP-1 dependent recruitment of the amyotrophic lateral sclerosis-associated protein FUS/TLS to sites of oxidative DNA damageStuart L Rulten, Amy Rotheray, Ryan L Green, et al.
Neurology. Genetics|August 16, 2018
Confirming TDP2 mutation in spinocerebellar ataxia autosomal recessive 23 (SCAR23)Guido Zagnoli-Vieira, Francesco Bruni, Kyle Thompson, et al.
Nucleic Acids Research|June 29, 2012
TDP2 promotes repair of topoisomerase I-mediated DNA damage in the absence of TDP1Zhihong Zeng, Abhishek Sharma, Limei Ju, et al.
Cell Reports|January 17, 2019
Efficient Single-Strand Break Repair Requires Binding to Both Poly(ADP-Ribose) and DNA by the Central BRCT Domain of XRCC1Luis M Polo, Yingqi Xu, Peter Hornyak, et al.
Molecular Cell|January 8, 2011
PARP-3 and APLF function together to accelerate nonhomologous end-joiningStuart L Rulten, Anna E O Fisher, Isabelle Robert, et al.
Life Science Alliance|February 28, 2019
FUS (fused in sarcoma) is a component of the cellular response to topoisomerase I-induced DNA breakage and transcriptional stressMaria Isabel Martinez-Macias, Duncan Aq Moore, Ryan L Green, et al.
EMBO Reports|May 1, 2021
Parp1 hyperactivity couples DNA breaks to aberrant neuronal calcium signalling and lethal seizuresEmilia Komulainen, Jack Badman, Stephanie Rey, et al.
Nature|September 3, 2025
DNA2 enables growth by restricting recombination-restarted replicationJessica J R Hudson, Rowin Appanah, David Jones, et al.
DNA Repair|September 24, 2004
The ataxia-oculomotor apraxia 1 gene product has a role distinct from ATM and interacts with the DNA strand break repair proteins XRCC1 and XRCC4Paula M Clements, Claire Breslin, Emma D Deeks, et al.
Pageof 12