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Annual Review of Medicine|October 9, 2007
A role for JAK2 mutations in myeloproliferative diseasesKelly J Morgan, D Gary GillilandBest Practice & Research. Clinical Haematology|August 26, 2003
FLT3-activating mutations in acute promyelocytic leukaemia: a rationale for risk-adapted therapy with FLT3 inhibitorsD Gary GillilandSeminars in Hematology|November 26, 2002
Molecular genetics of human leukemias: new insights into therapyD Gary GillilandMayo Clinic Proceedings|July 13, 2005
The JAK2V617F tyrosine kinase mutation in myeloproliferative disorders: status report and immediate implications for disease classification and diagnosisAyalew Tefferi, D Gary GillilandCell Cycle (Georgetown, Tex.)|March 14, 2007
Oncogenes in myeloproliferative disordersAyalew Tefferi, D Gary GillilandCell Cycle (Georgetown, Tex.)|June 23, 2005
JAK2 in myeloproliferative disorders is not just another kinaseAyalew Tefferi, D Gary GillilandCell Stem Cell|March 7, 2009
A radical bailout strategy for cancer stem cellsZuzana Tothova, D Gary GillilandCancer Cell|October 7, 2008
Going with the flow: JAK-STAT signaling in JMMLDemetrios Kalaitzidis, D Gary GillilandCell Stem Cell|March 29, 2008
FoxO transcription factors and stem cell homeostasis: insights from the hematopoietic systemZuzana Tothova, D Gary GillilandPageof 18