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Proceedings of the National Academy of Sciences of the United States of America|April 21, 2017
Unifying mechanism for different fibrotic diseasesGerlinde Wernig, Shih-Yu Chen, Lu Cui, et al.The Journal of Clinical Investigation|February 18, 2004
Conditional expression of oncogenic K-ras from its endogenous promoter induces a myeloproliferative diseaseIris T Chan, Jeffery L Kutok, Ifor R Williams, et al.Blood|March 4, 2010
The Apc(min) mouse has altered hematopoietic stem cell function and provides a model for MPD/MDSSteven W Lane, Stephen M Sykes, Fatima Al-Shahrour, et al.Stem Cells (Dayton, Ohio)|May 28, 2010
Hematopoietic stem cell defects in mice with deficiency of Fancd2 or Usp1Kalindi Parmar, Jungmin Kim, Stephen M Sykes, et al.Blood|February 24, 2006
Prospective analysis of TEL/AML1-positive patients treated on Dana-Farber Cancer Institute Consortium Protocol 95-01Mignon L Loh, Meredith A Goldwasser, Lewis B Silverman, et al.Cancer Cell|April 5, 2003
MOZ-TIF2-induced acute myeloid leukemia requires the MOZ nucleosome binding motif and TIF2-mediated recruitment of CBPKenji Deguchi, Paul M Ayton, Melina Carapeti, et al.Cancer Cell|December 21, 2004
MOZ-TIF2, but not BCR-ABL, confers properties of leukemic stem cells to committed murine hematopoietic progenitorsBrian J P Huntly, Hirokazu Shigematsu, Kenji Deguchi, et al.Nature|November 13, 2009
Direct inhibition of the NOTCH transcription factor complexRaymond E Moellering, Melanie Cornejo, Tina N Davis, et al.Leukemia Research|January 16, 2007
The GAP-related domain of neurofibromin attenuates proliferation and downregulates N- and K-Ras activation in Nf1-negative AML cellsKelly J Morgan, Matthew A Rowley, Stephen M Wiesner, et al.The Journal of Clinical Investigation|March 10, 2007
The homeobox gene CDX2 is aberrantly expressed in most cases of acute myeloid leukemia and promotes leukemogenesisClaudia Scholl, Dimple Bansal, Konstanze Döhner, et al.Pageof 18