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British Journal of Haematology|October 18, 2005
The JAK2(V617F) tyrosine kinase mutation in myelofibrosis with myeloid metaplasia: lineage specificity and clinical correlatesAyalew Tefferi, Terra L Lasho, Susan M Schwager, et al.Proceedings of the National Academy of Sciences of the United States of America|February 21, 2004
Combination of rapamycin and protein tyrosine kinase (PTK) inhibitors for the treatment of leukemias caused by oncogenic PTKsM Golam Mohi, Christina Boulton, Ting-Lei Gu, et al.British Journal of Haematology|October 4, 2005
JAK2 mutation in essential thrombocythaemia: clinical associations and long-term prognostic relevanceAlexandra P Wolanskyj, Terra L Lasho, Susan M Schwager, et al.Cell Stem Cell|September 13, 2008
Notch signaling specifies megakaryocyte development from hematopoietic stem cellsThomas Mercher, Melanie G Cornejo, Christopher Sears, et al.Blood|October 31, 2002
The protein tyrosine kinase inhibitor SU5614 inhibits FLT3 and induces growth arrest and apoptosis in AML-derived cell lines expressing a constitutively activated FLT3Karsten Spiekermann, Ralf J Dirschinger, Ruth Schwab, et al.Blood|April 30, 2005
The JAK2 V617F activating tyrosine kinase mutation is an infrequent event in both "atypical" myeloproliferative disorders and myelodysplastic syndromesDavid P Steensma, Gordon W Dewald, Terra L Lasho, et al.Leukemia|October 22, 2004
KIAA1509 is a novel PDGFRB fusion partner in imatinib-responsive myeloproliferative disease associated with a t(5;14)(q33;q32)R L Levine, M Wadleigh, D W Sternberg, et al.Blood|July 30, 2008
K-RasG12D-induced T-cell lymphoblastic lymphoma/leukemias harbor Notch1 mutations and are sensitive to gamma-secretase inhibitorsThomas Kindler, Melanie G Cornejo, Claudia Scholl, et al.Proceedings of the National Academy of Sciences of the United States of America|June 13, 2002
PML/RARalpha and FLT3-ITD induce an APL-like disease in a mouse modelLouise M Kelly, Jeffrey L Kutok, Ifor R Williams, et al.Proceedings of the National Academy of Sciences of the United States of America|April 12, 2006
Absence of the transcription factor CCAAT enhancer binding protein alpha results in loss of myeloid identity in bcr/abl-induced malignancyKatharina Wagner, Pu Zhang, Frank Rosenbauer, et al.Pageof 18