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Kelly M Ramsbottom

Showing results (21-30 of 25) with videos related to

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Cancer Immunology Research|December 11, 2020
Combined BRAF, MEK, and CDK4/6 Inhibition Depletes Intratumoral Immune-Potentiating Myeloid Populations in MelanomaEmily J Lelliott, Stefano Mangiola, Kelly M Ramsbottom, et al.
Cancer Immunology Research|November 16, 2021
BET Inhibition Enhances TNF-Mediated Antitumor ImmunityLisa C Wellinger, Simon J Hogg, Dane M Newman, et al.
Cancer Discovery|May 15, 2021
CDK4/6 Inhibition Promotes Antitumor Immunity through the Induction of T-cell MemoryEmily J Lelliott, Isabella Y Kong, Magnus Zethoven, et al.
Nature Communications|May 14, 2021
Antigen-driven EGR2 expression is required for exhausted CD8<sup>+</sup> T cell stability and maintenanceMayura V Wagle, Stephin J Vervoort, Madison J Kelly, et al.
Immunity|May 22, 2024
The CD8<sup>+</sup> T cell tolerance checkpoint triggers a distinct differentiation state defined by protein translation defectsWillem Van Der Byl, Simone Nüssing, Timothy J Peters, et al.
Pageof 3

Showing results (21-30 of 25) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 25 results.
Cancer Immunology Research|December 11, 2020
Combined BRAF, MEK, and CDK4/6 Inhibition Depletes Intratumoral Immune-Potentiating Myeloid Populations in MelanomaEmily J Lelliott, Stefano Mangiola, Kelly M Ramsbottom, et al.
Cancer Immunology Research|November 16, 2021
BET Inhibition Enhances TNF-Mediated Antitumor ImmunityLisa C Wellinger, Simon J Hogg, Dane M Newman, et al.
Cancer Discovery|May 15, 2021
CDK4/6 Inhibition Promotes Antitumor Immunity through the Induction of T-cell MemoryEmily J Lelliott, Isabella Y Kong, Magnus Zethoven, et al.
Nature Communications|May 14, 2021
Antigen-driven EGR2 expression is required for exhausted CD8<sup>+</sup> T cell stability and maintenanceMayura V Wagle, Stephin J Vervoort, Madison J Kelly, et al.
Immunity|May 22, 2024
The CD8<sup>+</sup> T cell tolerance checkpoint triggers a distinct differentiation state defined by protein translation defectsWillem Van Der Byl, Simone Nüssing, Timothy J Peters, et al.
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