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Experimental Neurology|October 6, 2023
Alterations in neurotransmitter co-release in Parkinson's diseaseKelsey Barcomb, Christopher P FordMolecular Pharmacology|September 24, 2013
Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2BKelsey Barcomb, Steven J Coultrap, K Ulrich BayerPlos One|May 23, 2012
A significant but rather mild contribution of T286 autophosphorylation to Ca2+/CaM-stimulated CaMKII activitySteven J Coultrap, Kelsey Barcomb, K Ulrich BayerCell Reports|July 25, 2019
Cholinergic Transmission at Muscarinic Synapses in the Striatum Is Driven Equally by Cortical and Thalamic InputsAphroditi A Mamaligas, Kelsey Barcomb, Christopher P FordThe Journal of Biological Chemistry|June 2, 2016
The CaMKII/GluN2B Protein Interaction Maintains Synaptic StrengthKelsey Barcomb, Johannes W Hell, Tim A Benke, et al.The Journal of Physiology|September 26, 2022
Properties and modulation of excitatory inputs to the locus coeruleusKelsey Barcomb, Samantha S Olah, Matthew J Kennedy, et al.The European Journal of Neuroscience|February 27, 2018
Synaptic function and plasticity in identified inhibitory inputs onto VTA dopamine neuronsAbigail M Polter, Kelsey Barcomb, Ayumi C Tsuda, et al.Journal of Neurochemistry|July 28, 2015
Live imaging of endogenous Ca²⁺/calmodulin-dependent protein kinase II in neurons reveals that ischemia-related aggregation does not require kinase activityKelsey Barcomb, Dayton J Goodell, Don B Arnold, et al.Neuropharmacology|December 28, 2010
A systems genetic analysis of alcohol drinking by mice, rats and men: influence of brain GABAergic transmissionLaura M Saba, Beth Bennett, Paula L Hoffman, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 21, 2014
Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strengthKelsey Barcomb, Isabelle Buard, Steven J Coultrap, et al.Pageof 2