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Toxins|January 19, 2011
Host-Toxin Interactions Involving EspC and Pet, Two Serine Protease Autotransporters of the EnterobacteriaceaeFernando Navarro-Garcia, Michael Sonnested, Ken TeterInfection and Immunity|September 19, 2019
Bimodal Response to Shiga Toxin 2 Subtypes Results from Relatively Weak Binding to the Target CellPatrick Cherubin, Dennis Fidler, Beatriz Quiñones, et al.Infection and Immunity|November 17, 2020
cAMP-Independent Activation of the Unfolded Protein Response by Cholera ToxinTuhina Banerjee, Aby Grabon, Michael Taylor, et al.Traffic (Copenhagen, Denmark)|April 16, 2003
A class of mutant CHO cells resistant to cholera toxin rapidly degrades the catalytic polypeptide of cholera toxin and exhibits increased endoplasmic reticulum-associated degradationKen Teter, Michael G Jobling, Randall K HolmesInfection and Immunity|November 24, 2004
Vesicular transport is not required for the cytoplasmic pool of cholera toxin to interact with the stimulatory alpha subunit of the heterotrimeric g proteinKen Teter, Michael G Jobling, Randall K HolmesJournal of Visualized Experiments : Jove|January 11, 2012
Detection of toxin translocation into the host cytosol by surface plasmon resonanceMichael Taylor, Tuhina Banerjee, Neyda VanBennekom, et al.Cellular Signalling|November 12, 2022
The cytopathic activity of cholera toxin requires a threshold quantity of cytosolic toxinCarly Bader, Michael Taylor, Tuhina Banerjee, et al.Frontiers in Cellular and Infection Microbiology|November 8, 2021
Proline Isomerization as a Key Determinant for Hsp90-Toxin InteractionsAlisha Kellner, Patrick Cherubin, James K Harper, et al.Biochemistry|August 9, 2019
Stability and Conformational Resilience of Protein Disulfide IsomeraseJessica Guyette, Baggio Evangelista, Suren A Tatulian, et al.Archives of Biochemistry and Biophysics|March 14, 2025
Amyloid β fragments that suppress oligomers but not fibrils are cytoprotectiveMaria C Zabala-Rodriguez, Ken Teter, Suren A TatulianPageof 7