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The American Journal of Clinical Nutrition|September 30, 2011
Soy isoflavone phase II metabolism differs between rodents and humans: implications for the effect on breast cancer riskKenneth D R Setchell, Nadine M Brown, Xueheng Zhao, et al.
Pediatric Blood & Cancer|April 12, 2012
Parent-child communication and psychological adjustment in children with a brain tumorAnnarita Adduci, Momcilo Jankovic, Sandra Strazzer, et al.
Metabolomics : Official Journal of the Metabolomic Society|July 1, 2026
Lipid reprogramming of stratified squamous epithelium by the high-risk HPV E6 and E6/E7 oncoproteinsMegan E Spurgeon, Taylor E Lange, Alexandra Baty, et al.
The Journal of Steroid Biochemistry and Molecular Biology|June 24, 2014
Synthesis of atypical bile acids for use as investigative tools for the genetic defect of 3β-hydroxy-Δ(5)-C27-steroid oxidoreductase deficiencyAntimo Gioiello, Bruno Cerra, Wujuan Zhang, et al.
The American Journal of Clinical Nutrition|July 30, 2002
Evidence for lack of absorption of soy isoflavone glycosides in humans, supporting the crucial role of intestinal metabolism for bioavailabilityKenneth D R Setchell, Nadine M Brown, Linda Zimmer-Nechemias, et al.
Cells|September 28, 2021
Substrate Reduction Therapy Reverses Mitochondrial, mTOR, and Autophagy Alterations in a Cell Model of Gaucher DiseaseYanyan Peng, Benjamin Liou, Yi Lin, et al.
Gastroenterology|January 4, 2003
Liver disease caused by failure to racemize trihydroxycholestanoic acid: gene mutation and effect of bile acid therapyKenneth D R Setchell, James E Heubi, Kevin E Bove, et al.
Gastroenterology|October 1, 2003
Biliary diversion for progressive familial intrahepatic cholestasis: improved liver morphology and bile acid profileAmethyst C Kurbegov, Kenneth D R Setchell, Joel E Haas, et al.
The Journal of Nutrition|April 4, 2003
Bioavailability, disposition, and dose-response effects of soy isoflavones when consumed by healthy women at physiologically typical dietary intakesKenneth D R Setchell, Nadine Maynard Brown, Pankaj B Desai, et al.
Scientific Reports|April 5, 2019
Combination of acid β-glucosidase mutation and Saposin C deficiency in mice reveals Gba1 mutation dependent and tissue-specific disease phenotypeBenjamin Liou, Wujuan Zhang, Venette Fannin, et al.
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