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Chembiochem : a European Journal of Chemical Biology|March 13, 2024
Monitoring Anthracycline Cancer Drug-Nucleosome Interaction by NMR Using a Specific Isotope Labeling Approach for Nucleosomal DNAClara L van Emmerik, Vincenzo Lobbia, Jacques Neefjes, et al.
Nature Communications|June 11, 2016
Cholesterol and ORP1L-mediated ER contact sites control autophagosome transport and fusion with the endocytic pathwayRuud H Wijdeven, Hans Janssen, Leila Nahidiazar, et al.
Cancer Medicine|January 6, 2025
The Impact of Chemotherapy and Body Mass Index on Cancer-Related Fatigue in Colon Cancer Patients: A PROFILES-Registry StudyAnneke Kastelein, Floortje Mols, Laura Kervezee, et al.
Journal of Cell Science|August 20, 2009
Mimicking proteasomal release of polyglutamine peptides initiates aggregation and toxicityMarcel Raspe, Judith Gillis, Hilde Krol, et al.
Radiation Research|August 4, 2012
IL-1 generated subsequent to radiation-induced tissue injury contributes to the pathogenesis of radiodermatitisMatthew Janko, Fernando Ontiveros, T J Fitzgerald, et al.
Annual Review of Immunology|January 27, 2017
The Biology and Underlying Mechanisms of Cross-Presentation of Exogenous Antigens on MHC-I MoleculesFreidrich M Cruz, Jeff D Colbert, Elena Merino, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 7, 2006
Endoplasmic reticulum aminopeptidase 1 (ERAP1) trims MHC class I-presented peptides in vivo and plays an important role in immunodominanceIan A York, Michael A Brehm, Sophia Zendzian, et al.
The American Journal of Surgical Pathology|December 3, 2008
An oncofetal protein IMP3: a new molecular marker for the detection of esophageal adenocarcinoma and high-grade dysplasiaDi Lu, Poonam Vohra, Peigou G Chu, et al.
Molecular Endocrinology (Baltimore, Md.)|May 30, 2009
Resistance to antiestrogen arzoxifene is mediated by overexpression of cyclin D1Wilbert Zwart, Mariska Rondaij, Kees Jalink, et al.
Nature Medicine|June 19, 2007
Identification of a key pathway required for the sterile inflammatory response triggered by dying cellsChun-Jen Chen, Hajime Kono, Douglas Golenbock, et al.
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