Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Kenneth R Brittian

Showing results (41-50 of 49) with videos related to

Pageof 5
Sort By:
You have reached the last page of results.This site can display upto 49 results.
Circulation|September 2, 2017
Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac GrowthAndrew A Gibb, Paul N Epstein, Shizuka Uchida, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 23, 2020
Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarctionTimothy N Audam, Yibing Nong, Alex Tomlin, et al.
Basic Research in Cardiology|March 17, 2017
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.
Plos One|November 30, 2020
Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarctionSujith Dassanayaka, Kenneth R Brittian, Bethany W Long, et al.
Circulation. Heart Failure|April 26, 2014
Metabolomic analysis of pressure-overloaded and infarcted mouse heartsBrian E Sansbury, Angelica M DeMartino, Zhengzhi Xie, et al.
Research Square|March 31, 2025
TGFβ-activated kinase 1 signaling controls acquisition of the inflammatory fibroblast phenotype and regulates cardiac remodeling after myocardial infarctionDaniel C Nguyen, Jonah K Stephan, Robert E Brainard, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 17, 2021
Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overloadTimothy N Audam, Caitlin M Howard, Lauren F Garrett, et al.
Nature Communications|May 26, 2026
TAK1 drives inflammatory fibroblast acquisition and shapes myocardial infarction responses in male miceDaniel C Nguyen, Jonah K Stephan, Lianay Gutierrez Luque, et al.
Journal of Molecular and Cellular Cardiology|July 12, 2025
Pyruvate kinase splice variants in fibroblasts influence cardiac remodeling after myocardial infarction in male miceCollin K Wells, Daniel C Nguyen, Robert E Brainard, et al.
Pageof 5

Showing results (41-50 of 49) with videos related to

Sort By:
Pageof 5
You have reached the last page of results.This site can display upto 49 results.
Circulation|September 2, 2017
Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac GrowthAndrew A Gibb, Paul N Epstein, Shizuka Uchida, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 23, 2020
Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarctionTimothy N Audam, Yibing Nong, Alex Tomlin, et al.
Basic Research in Cardiology|March 17, 2017
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.
Plos One|November 30, 2020
Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarctionSujith Dassanayaka, Kenneth R Brittian, Bethany W Long, et al.
Circulation. Heart Failure|April 26, 2014
Metabolomic analysis of pressure-overloaded and infarcted mouse heartsBrian E Sansbury, Angelica M DeMartino, Zhengzhi Xie, et al.
Research Square|March 31, 2025
TGFβ-activated kinase 1 signaling controls acquisition of the inflammatory fibroblast phenotype and regulates cardiac remodeling after myocardial infarctionDaniel C Nguyen, Jonah K Stephan, Robert E Brainard, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 17, 2021
Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overloadTimothy N Audam, Caitlin M Howard, Lauren F Garrett, et al.
Nature Communications|May 26, 2026
TAK1 drives inflammatory fibroblast acquisition and shapes myocardial infarction responses in male miceDaniel C Nguyen, Jonah K Stephan, Lianay Gutierrez Luque, et al.
Journal of Molecular and Cellular Cardiology|July 12, 2025
Pyruvate kinase splice variants in fibroblasts influence cardiac remodeling after myocardial infarction in male miceCollin K Wells, Daniel C Nguyen, Robert E Brainard, et al.
Pageof 5