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Cell Metabolism|January 31, 2017
Store-Operated Ca<sup>2+</sup> Entry Controls Induction of Lipolysis and the Transcriptional Reprogramming to Lipid MetabolismMate Maus, Mario Cuk, Bindi Patel, et al.Novartis Foundation Symposium|July 8, 2005
A molecular dissection of lymphocyte unresponsiveness induced by sustained calcium signallingVigo Heissmeyer, Fernando Macián, Rajat Varma, et al.Nature Immunology|February 21, 2025
STIM1-mediated NFAT signaling synergizes with STAT1 to control T-bet expression and T<sub>H</sub>1 differentiationLi Zhong, Yin-Hu Wang, Sascha Kahlfuss, et al.Biorxiv : the Preprint Server for Biology|February 8, 2024
Regulatory T cells and IFN-γ-producing Th1 cells play a critical role in the pathogenesis of Sjögren's SyndromeYin-Hu Wang, Wenyi Li, Maxwell McDermott, et al.Nature Communications|April 20, 2022
Cavβ1 regulates T cell expansion and apoptosis independently of voltage-gated Ca<sup>2+</sup> channel functionSerap Erdogmus, Axel R Concepcion, Megumi Yamashita, et al.Plos One|March 4, 2014
Store-operated Ca2+ entry (SOCE) regulates melanoma proliferation and cell migrationMasanari Umemura, Erdene Baljinnyam, Stefan Feske, et al.Nature Immunology|February 2, 2022
The volume-regulated anion channel LRRC8C suppresses T cell function by regulating cyclic dinucleotide transport and STING-p53 signalingAxel R Concepcion, Larry E Wagner, Jingjie Zhu, et al.Science Translational Medicine|December 18, 2024
IFN-γ-producing T<sub>H</sub>1 cells and dysfunctional regulatory T cells contribute to the pathogenesis of Sjögren's diseaseYin-Hu Wang, Wenyi Li, Maxwell McDermott, et al.The Journal of Allergy and Clinical Immunology|December 17, 2009
ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasiaChristie-Ann McCarl, Capucine Picard, Sara Khalil, et al.The Journal of Allergy and Clinical Immunology|November 21, 2017
ORAI1 mutations abolishing store-operated Ca<sup>2+</sup> entry cause anhidrotic ectodermal dysplasia with immunodeficiencyJayson Lian, Mario Cuk, Sascha Kahlfuss, et al.Pageof 14