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Genes & Development|July 14, 2010
Antisense correction of SMN2 splicing in the CNS rescues necrosis in a type III SMA mouse modelYimin Hua, Kentaro Sahashi, Gene Hung, et al.
Nucleic Acids Research|August 30, 2007
In vitro and in silico analysis reveals an efficient algorithm to predict the splicing consequences of mutations at the 5' splice sitesKentaro Sahashi, Akio Masuda, Tohru Matsuura, et al.
Nucleic Acids Research|April 20, 2018
Antisense oligonucleotides correct the familial dysautonomia splicing defect in IKBKAP transgenic miceRahul Sinha, Young Jin Kim, Tomoki Nomakuchi, et al.
Disease Models & Mechanisms|June 6, 2025
Label-free morphology-based phenotypic analysis of spinal and bulbar muscular atrophy muscle cell modelsKenji Sakakibara, Kenjiro Tanaka, Madoka Iida, et al.
Human Molecular Genetics|August 2, 2015
Silencing neuronal mutant androgen receptor in a mouse model of spinal and bulbar muscular atrophyKentaro Sahashi, Masahisa Katsuno, Gene Hung, et al.
Annals of Clinical and Translational Neurology|December 28, 2023
Clinicopathological features of graft versus host disease-associated myositisTomoyuki Kazuta, Ayuka Murakami, Seiya Noda, et al.
Molecular Therapy. Nucleic Acids|January 19, 2026
An acyclic nucleic acid-modified siRNA targeting CAG expansions for polyglutamine disease treatmentKentaro Maeda, Tomoki Hirunagi, Kentaro Sahashi, et al.
Journal of Cachexia, Sarcopenia and Muscle|November 8, 2023
Exercise attenuates polyglutamine-mediated neuromuscular degeneration in a mouse model of spinal and bulbar muscular atrophyTomoki Hirunagi, Hideaki Nakatsuji, Kentaro Sahashi, et al.
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