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Frontiers in Physiology|January 23, 2024
Enlarged colony housing promotes linear progression of subchondral bone remodeling in joint instability rat modelsStephanie Menges, Kerstin Kleinschmidt-Dörr, Christian Brenneis
Frontiers in Physiology|July 12, 2023
Anti-NGF treatment worsens subchondral bone and cartilage measures while improving symptoms in floor-housed rabbits with osteoarthritisStephanie Menges, Martin Michaelis, Kerstin Kleinschmidt-Dörr
Heliyon|September 15, 2021
Dynamic weight-bearing test during jumping: A sensitive outcome measure of chronic osteoarthritis pain in ratsAndreas Westhof, Kerstin Kleinschmidt-Doerr, Martin Michaelis, et al.
Acta Biomaterialia|February 4, 2010
Chondrogenic pre-induction of human mesenchymal stem cells on beta-TCP: enhanced bone quality by endochondral heterotopic bone formationPatricia Janicki, Philip Kasten, Kerstin Kleinschmidt, et al.
Journal of Cellular and Molecular Medicine|June 5, 2020
The GDF-5 mutant M1673 exerts robust anabolic and anti-catabolic effects in chondrocytesTanja Mang, Kerstin Kleinschmidt-Dörr, Frank Ploeger, et al.
The Journal of Bone and Joint Surgery. American Volume|October 17, 2014
Superior angiogenic potential of GDF-5 and GDF-5(V453/V456) compared with BMP-2 in a rabbit long-bone defect modelKerstin Kleinschmidt, Mechthild Wagner-Ecker, Benjamin Bartek, et al.
Biomaterials|May 18, 2013
Enhanced reconstruction of long bone architecture by a growth factor mutant combining positive features of GDF-5 and BMP-2Kerstin Kleinschmidt, Frank Ploeger, Joachim Nickel, et al.
Acta Orthopaedica|June 28, 2016
Increased bone formation in a rabbit long-bone defect model after single local and single systemic application of erythropoietinGeorg W Omlor, Kerstin Kleinschmidt, Simone Gantz, et al.
Acta Biomaterialia|March 26, 2015
Mesenchymal stromal cell implantation for stimulation of long bone healing aggravates Staphylococcus aureus induced osteomyelitisElisabeth Seebach, Jeannine Holschbach, Nicole Buchta, et al.
Journal of Cell Science|August 9, 2020
BMPR1A is necessary for chondrogenesis and osteogenesis, whereas BMPR1B prevents hypertrophic differentiationTanja Mang, Kerstin Kleinschmidt-Doerr, Frank Ploeger, et al.
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