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Kidney International|August 9, 2013
Podocyte-specific deletion of NDST1, a key enzyme in the sulfation of heparan sulfate glycosaminoglycans, leads to abnormalities in podocyte organization in vivoTerrel Sugar, Deborah J Wassenhove-McCarthy, Jeffrey D Esko, et al.American Journal of Physiology. Renal Physiology|March 4, 2016
N-sulfation of heparan sulfate is critical for syndecan-4-mediated podocyte cell-matrix interactionsTerrel Sugar, Deborah J Wassenhove-McCarthy, A Wayne Orr, et al.Plos One|January 12, 2017
p62 Pathology Model in the Rat Substantia Nigra with Filamentous Inclusions and Progressive NeurodegenerationKasey L Jackson, Wen-Lang Lin, Sumitra Miriyala, et al.Redox Biology|October 6, 2017
Cardiac-specific inactivation of LPP3 in mice leads to myocardial dysfunction and heart failureMini Chandra, Diana Escalante-Alcalde, Md Shenuarin Bhuiyan, et al.Kidney International|May 16, 2008
Loss of heparan sulfate glycosaminoglycan assembly in podocytes does not lead to proteinuriaShoujun Chen, Deborah J Wassenhove-McCarthy, Yu Yamaguchi, et al.Redox Biology|October 13, 2020
SOD2 deficiency in cardiomyocytes defines defective mitochondrial bioenergetics as a cause of lethal dilated cardiomyopathySudha Sharma, Susmita Bhattarai, Hosne Ara, et al.Current Opinion in Molecular Therapeutics|August 19, 2007
Anti-proteinuric effects of glycosaminoglycan-based drugsTessa J M Wijnhoven, Joost F M Lensen, Angelique L Rops, et al.Human Molecular Genetics|February 2, 2011
Mutations in PPIB (cyclophilin B) delay type I procollagen chain association and result in perinatal lethal to moderate osteogenesis imperfecta phenotypesShawna M Pyott, Ulrike Schwarze, Helena E Christiansen, et al.Pageof 2