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Mbio|September 25, 2023
Nectin-3 and shed forms of CSPG4 can serve as epithelial cell receptors for Clostridioides difficile TcdBKevin O Childress, Caroline S Cencer, Matthew J Tyska, et al.
Plos Pathogens|August 21, 2018
The C. difficile clnRAB operon initiates adaptations to the host environment in response to LL-37Emily C Woods, Adrianne N Edwards, Kevin O Childress, et al.
Infection and Immunity|September 21, 2016
The Phosphotransfer Protein CD1492 Represses Sporulation Initiation in Clostridium difficileKevin O Childress, Adrianne N Edwards, Kathryn L Nawrocki, et al.
Anaerobe|January 11, 2015
An alkaline phosphatase reporter for use in Clostridium difficileAdrianne N Edwards, Ricardo A Pascual, Kevin O Childress, et al.
Plos Pathogens|February 17, 2022
Glucosyltransferase-dependent and independent effects of Clostridioides difficile toxins during infectionF Christopher Peritore-Galve, John A Shupe, Rory J Cave, et al.
Biorxiv : the Preprint Server for Biology|May 27, 2024
Inhibition of EGFR/ErbB does not protect against C. difficile toxin BUswah Siddiqi, Hannah M Lunnemann, Kevin O Childress, et al.
Infection and Immunity|January 20, 2021
Murine Intrarectal Instillation of Purified Recombinant Clostridioides difficile Toxins Enables Mechanistic Studies of PathogenesisNicholas O Markham, Sarah C Bloch, John A Shupe, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 22, 2020
Clostridioides difficile infection damages colonic stem cells via TcdB, impairing epithelial repair and recovery from diseaseSteven J Mileto, Thierry Jardé, Kevin O Childress, et al.
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