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Epilepsia|November 8, 2014
Seizure expression, behavior, and brain morphology differences in colonies of Genetic Absence Epilepsy Rats from StrasbourgKim L Powell, Howard Tang, Caroline Ng, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 16, 2009
A Cav3.2 T-type calcium channel point mutation has splice-variant-specific effects on function and segregates with seizure expression in a polygenic rat model of absence epilepsyKim L Powell, Stuart M Cain, Caroline Ng, et al.Neurology|July 31, 2012
Glutamate is associated with a higher risk of seizures in patients with gliomasTanya I Yuen, Andrew P Morokoff, Andrew Bjorksten, et al.Neurobiology of Disease|September 14, 2021
Altered cardiac structure and function is related to seizure frequency in a rat model of chronic acquired temporal lobe epilepsyKim L Powell, Zining Liu, Claire L Curl, et al.Epilepsia|March 6, 2014
HCN channelopathy and cardiac electrophysiologic dysfunction in genetic and acquired rat epilepsy modelsKim L Powell, Nigel C Jones, Jeremy T Kennard, et al.Epilepsia|February 23, 2018
CaV 3.2 drives sustained burst-firing, which is critical for absence seizure propagation in reticular thalamic neuronsStuart M Cain, John R Tyson, Hyun-Beom Choi, et al.Science Translational Medicine|February 21, 2012
T-type calcium channel blockers that attenuate thalamic burst firing and suppress absence seizuresElizabeth Tringham, Kim L Powell, Stuart M Cain, et al.Journal of Molecular and Cellular Cardiology|March 14, 2024
Myocardial glycophagy flux dysregulation and glycogen accumulation characterize diabetic cardiomyopathyKimberley M Mellor, Upasna Varma, Parisa Koutsifeli, et al.Pageof 4